Activation of c-Jun N-terminal kinase (JNK) by widely used specific p38 MAPK inhibitors SB202190 and SB203580: A MLK-3-MKK7-dependent mechanism
SB203580 increases G-CSF production via a stem-loop destabilizing element in the 3’ untranslated region in macrophages independently of its effect on p38 MAPK activity
Phosphorylation of initiation factor 4E is resistant to SB203580 in cells expressing a drug-resistant mutant of stress-activated protein kinase 2a/p38
Blockade by SB203580 ofCyp1a1Induction by 2,3,7,8-Tetrachlorodibenzo-p-dioxin, and the Possible Mechanism: Possible Involvement of the p38 Mitogen-Activated Protein Kinase Pathway in Shuttling of Ah Receptor Overexpressed in COS-7 Cells
