Results 271 to 280 of about 213,259 (308)
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Effects of short chain fatty acids on seedlings

Plant, Cell and Environment, 1982
Abstract. Primary roots of lettuce show no appreciable diminution of sensitivity of SCFA between 24 and 72 h, so it is likely that all actively growing primary roots are susceptible to inhibition by SCFA. While roots do not recover from long exposures to high concentrations of SCFA, partial recovery is seen following exposure to intermediate levels.
Corinne E. Ulbright   +2 more
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Effects of Short-Chain Fatty Acids on Gastrointestinal Motility

Scandinavian Journal of Gastroenterology, 1997
Besides their action on gut morphology and function, short-chain fatty acids (SCFAs), produced by bacterial fermentation of carbohydrates in the colon, influence gastrointestinal motility. As they are not present in the stomach and proximal small intestine, SCFAs do not directly affect motility of these segments.
Cherbut, Christine   +3 more
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Transport of Short Chain Fatty Acids

1974
The present interest in the mechanisms involved in intestinal transport of short-chain fatty acids (SCFA) derives from the experiments of Smyth and Taylor [1,2] which showed that compounds of this type were transported against gradients of concentration from luminal to serosal sides of rat intestine incubated in vitro.
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Absorption and metabolism of short‐chain fatty acids in ruminants

Archiv für Tierernaehrung, 1998
Short-chain fatty acids (SCFA), viz. acetate, propionate and butyrate are quantitatively important substrates in ruminant energy metabolism. In the reviewed literature, 16 44% of ME intake was recovered as portal appearance of SCFA. This is considerably lower than expected when related to the estimated intragastric flux of SCFA.
Kristensen, Niels Bastian; id_orcid 0000-0003-0214-2929   +2 more
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Short-Chain Fatty Acids in Ulcerative Colitis

Nutrition Reviews, 2009
Short-chain fatty acids (SCFA), a by-product of anaerobic fermentation of undigested carbohydrates within the colon, are an important energy source for colonic epithelial cells. It has been proposed that ulcerative colitis, a chronic inflammatory condition of the colon, may result from a metabolic defect in SCFA oxidation in the colon and that this ...
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Metabolic and Intestinal Effects of Short‐Chain Fatty Acids

Journal of Parenteral and Enteral Nutrition, 1990
Current research has identified the metabolic and intestinal effects of SCFA in experimental models and humans. Ample physiologic rationale has come forth to justify controlled studies of the administration of SCFA in selected patients with intestinal dysfunction.
J L, Rombeau, S A, Kripke
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Short-chain fatty acid, acylation and cardiovascular diseases

Clinical Science, 2020
Abstract Cardiovascular diseases (CVDs) are the leading cause of morbidity and mortality worldwide. Metabolic dysfunction is a fundamental core mechanism underlying CVDs. Previous studies generally focused on the roles of long-chain fatty acids (LCFAs) in CVDs.
Xiao-Feng Chen   +2 more
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Short-Chain Fatty Acids in Germfree Mice and Rats

The Journal of Nutrition, 1986
The short-chain fatty acids (SCFAs) have been analyzed in small intestinal and cecal content of 10 germfree and six conventional mice from a Norwegian laboratory, in cecal content and serum of five germfree rats and two germfree mice from a Swedish laboratory and in Norwegian and Swedish autoclaved, nonpurified rodor diets.
T, Høverstad, T, Midtvedt
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Short Chain Fatty Acids

1997
Short chain fatty acids (SCFA) are produced in the human colon by bacterial fermentation of carbohydrates and amino acids. The physiological importance of these molecules is increasingly recognised. They have been shown to influence cellular turnover not only in the colon, where they are formed, but also at distant sites in the small intestine (Sakata ...
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Regulation of Insulin Secretion by Short Chain Fatty Acids

Nature, 1968
EVIDENCE is accumulating that in some mammalian species there may be a direct effect of short chain fatty acids on the secretion of insulin from the pancreas. Thus it has been shown that octanoate causes a marked release of insulin from slices of rat pancreas1.
W, Montague, K W, Taylor
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