Results 301 to 310 of about 165,371 (351)
CD109 Deletion Promotes Myofibroblast Differentiation and Smad-Dependent Matrix Accumulation in Skin Fibrosis. [PDF]
Int J Mol SciXu L +4 more
europepmc +1 more source
Interplay between TGF-β signaling and long non-coding RNAs in digestive system cancers: mechanisms and biological implications. [PDF]
Hum CellLi P, Huang D, Gu X.
europepmc +1 more source
miR-1737 targets TAK1 to mediate the BMP-Smad signaling pathway to regulate the molecular mechanism of chicken tibial chondrodysplasia. [PDF]
Poult SciXu H +10 more
europepmc +1 more source
SULT1E1 suppresses hepatocellular carcinoma progression via activation of the TGF-β/SMAD signaling pathway and cell cycle arrest. [PDF]
BMC GastroenterolZhang W +5 more
europepmc +1 more source
Some of the next articles are maybe not open access.
Related searches:
Related searches:
Proceedings of the 30th ACM International Conference on Information & Knowledge Management, 2021
Ad retrieval in sponsored search aims to understand user search intentions (user queries) and retrieves a set of ads inferred as being relevant to the queries. Due to the huge amount of search traffic and multiple views of relevance (such as co-clicking, co-bidding or textual similar), it is highly desirable but remain challenging to achieve a large ...
Shiyang Wen +6 more
openaire +1 more source
Ad retrieval in sponsored search aims to understand user search intentions (user queries) and retrieves a set of ads inferred as being relevant to the queries. Due to the huge amount of search traffic and multiple views of relevance (such as co-clicking, co-bidding or textual similar), it is highly desirable but remain challenging to achieve a large ...
Shiyang Wen +6 more
openaire +1 more source
A Smad Transcriptional Corepressor [PDF]
Cell, 1999 Following TGFbeta receptor-mediated phosphorylation and association with Smad4, Smad2 moves into the nucleus, binds to target promoters in association with DNA-binding cofactors, and recruits coactivators such as p300/CBP to activate transcription. We identified the homeodomain protein TGIF as a Smad2-binding protein and a repressor of transcription. A
David Wotton +2 more
exaly +3 more sources
The International Journal of Biochemistry & Cell Biology, 1999
The Smads are a family of intracellular signalling molecules that act downstream of receptors for the transforming growth factor (TGF)-beta family of ligands. Three classes of Smads have been identified. The receptor-regulated Smads are direct substrates for the type I receptors, which are serine/threonine kinases.
openaire +2 more sources
The Smads are a family of intracellular signalling molecules that act downstream of receptors for the transforming growth factor (TGF)-beta family of ligands. Three classes of Smads have been identified. The receptor-regulated Smads are direct substrates for the type I receptors, which are serine/threonine kinases.
openaire +2 more sources
Science Signaling, 2000
The transforming growth factor–β (TGF-β) superfamily of secreted polypeptide growth factors exerts extensive control over all aspects of development and homeostasis, and components of this pathway are often mutated in cancers and in several hereditary disorders.
openaire +2 more sources
The transforming growth factor–β (TGF-β) superfamily of secreted polypeptide growth factors exerts extensive control over all aspects of development and homeostasis, and components of this pathway are often mutated in cancers and in several hereditary disorders.
openaire +2 more sources
Smad-dependent and Smad-independent pathways in TGF-β family signalling
Nature, 2003Transforming growth factor-beta (TGF-beta) proteins regulate cell function, and have key roles in development and carcinogenesis. The intracellular effectors of TGF-beta signalling, the Smad proteins, are activated by receptors and translocate into the nucleus, where they regulate transcription. Although this pathway is inherently simple, combinatorial
Ying E Zhang
exaly +3 more sources