Results 101 to 110 of about 65,066 (300)

Clumps in stellar winds [PDF]

open access: yesASTRA Proceedings, 2014
Abstract. We discuss the origin and quantification of wind clumping and mass–loss rates (Ṁ), particularly in close proximity to the Eddington (Γ) limit, relevant for very massive stars (VMS). We present evidence that clumping may not be the result of the line-deshadowing instability (LDI), but that clumps are already present in the stellar photosphere.
openaire   +2 more sources

Targeted modulation of IGFL2‐AS1 reveals its translational potential in cervical adenocarcinoma

open access: yesMolecular Oncology, EarlyView.
Cervical adenocarcinoma patients face worse outcomes than squamous cell carcinoma counterparts despite similar treatment. The identification of IGFL2‐AS1's differential expression provides a molecular basis for distinguishing these histotypes, paving the way for personalized therapies and improved survival in vulnerable populations globally.
Ricardo Cesar Cintra   +6 more
wiley   +1 more source

New insight into the physics of atmospheres of early type stars [PDF]

open access: yes
The phenomenon of mass loss and stellar winds from hot stars are discussed. The mass loss rate of early type stars increases by about a factor of 100 to 1000 during their evolution.
Lamers, H. J. G. L. M.
core   +1 more source

Stellar hydrodynamical modeling of dwarf galaxies: simulation methodology, tests, and first results

open access: yes, 2015
Cosmological simulations still lack numerical resolution or physical processes to simulate dwarf galaxies in sufficient details. Accurate numerical simulations of individual dwarf galaxies are thus still in demand.
Hensler, Gerhard   +2 more
core   +1 more source

Network divergence analysis identifies adaptive gene modules and two orthogonal vulnerability axes in pancreatic cancer

open access: yesMolecular Oncology, EarlyView.
Tumors contain diverse cellular states whose behavior is shaped by context‐dependent gene coordination. By comparing gene–gene relationships across biological contexts, we identify adaptive transcriptional modules that reorganize into distinct vulnerability axes.
Brian Nelson   +9 more
wiley   +1 more source

Molecular Bubble and Outflow in S Mon Revealed by Multiband Data Sets

open access: yesThe Astrophysical Journal
We identify a molecular bubble, and study the star formation and its feedback in the S Mon region, using multiple molecular lines, young stellar objects (YSOs), and infrared data.
Dejian Liu   +10 more
doaj   +1 more source

The impact of metallicity and dynamics on the evolution of young star clusters

open access: yes, 2014
The early evolution of a dense young star cluster (YSC) depends on the intricate connection between stellar evolution and dynamical processes. Thus, N-body simulations of YSCs must account for both aspects.
Bressan, A., Mapelli, M., Trani, A. A.
core  

RIPK4 function interferes with melanoma cell adhesion and metastasis

open access: yesMolecular Oncology, EarlyView.
RIPK4 promotes melanoma growth and spread. RIPK4 levels increase as skin lesions progress to melanoma. CRISPR/Cas9‐mediated deletion of RIPK4 causes melanoma cells to form less compact spheroids, reduces their migratory and invasive abilities and limits tumour growth and dissemination in mouse models.
Norbert Wronski   +9 more
wiley   +1 more source

Revisiting the evolutionary status of massive stars in the central parsec of the Milky Way

open access: yesAstronomy & Astrophysics
Context. Massive stars and their winds strongly affect their environment. For example, they determine the accretion rate on to the Galactic centre (GC) supermassive black hole Sagittarius A* (Sgr A*).
Gormaz-Matamala A. C.   +4 more
doaj   +1 more source

EDNRB‐dependent endothelin signaling reduces proliferation and promotes proneural‐to‐mesenchymal transition in gliomas

open access: yesMolecular Oncology, EarlyView.
Glioma cells mainly express the endothelin receptor EDNRB, while EDNRA is restricted to a perivascular tumor subpopulation. Endothelin signaling reduces glioma cell proliferation while promoting migration and a proneural‐to‐mesenchymal transition associated with poor prognosis. This pathway activates Ca2+, K+, ERK, and STAT3 signalings and is regulated
Donovan Pineau   +36 more
wiley   +1 more source

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