Results 181 to 190 of about 19,285 (227)

Negative Regulator of Interferon Response Promotes Toll-like Receptor 3-induced Interleukin 6 in Rheumatoid Synoviocyte. [PDF]

open access: yesIn Vivo
Wada K   +10 more
europepmc   +1 more source

Induction of Interleukin-23 p19 by Serum Amyloid a (SAA) in Rheumatoid Synoviocytes

open access: yesClinical and Experimental Immunology, 2010
Summary In this study, we investigated the roles of serum amyloid A (SAA) in T helper 17 (Th17)-related cytokine induction in rheumatoid arthritis (RA) synoviocytes.
K Migita, T Miyashita, A Komori
exaly   +2 more sources

Potential role of mitochondria in synoviocytes

Clinical Rheumatology, 2020
Synoviocytes are located in the synovium lining layer, which is composed of macrophage-like synoviocytes (MLS) and fibroblast-like synoviocytes (FLS) with different characteristics. Mitochondria, which exist in most cells, are two membrane-covered organelles.
Muzhe, Li   +6 more
openaire   +2 more sources

Activation of synoviocytes

Current Opinion in Rheumatology, 2000
The evaluation of molecular pathways has revealed various novel insights into rheumatoid arthritis pathophysiology during the past year. In addition, there is an increasing tendency toward analysis not merely of a single mechanism but rather of data addressing a substantial part of the cascade of events leading to cellular activation.
U, Müller-Ladner, R E, Gay, S, Gay
openaire   +2 more sources

Ceria Nanoparticles Alleviated Osteoarthritis Through Attenuating Senescence and Senescence-Associated Secretory Phenotype in Synoviocytes

open access: yesInternational Journal of Molecular Sciences, 2023
Accumulation of senescent cells is the prominent risk factor for osteoarthritis (OA), accelerating the progression of OA through a senescence-associated secretory phenotype (SASP).
Pang-Hu Zhou   +2 more
exaly   +2 more sources

Wnt signaling in rheumatoid synoviocyte activation

Modern Rheumatology, 2002
Abstract Rheumatoid arthritis (RA) is a joint-specific disease with complex pathogenesis. It is characterized by synovial inflammation, cartilage loss, and joint destruction. The reasons why joint damage recurs when therapy is discontinued are not clearly understood. Several lines of evidence suggest that cartilage damage is promoted by the transformed
M, Sen, D A, Carson
openaire   +2 more sources

Early synovitis—Synoviocytes and mononuclear cells

Seminars in Arthritis and Rheumatism, 1994
Immunohistological observations of synovial tissues obtained at the onset of rheumatoid arthritis (RA) are limited and often reflect the diagnostic confusion inherent in any study of early synovitis. However, a critical analysis of published information and the authors' experience allow certain preliminary conclusions.
N J, Zvaifler, D, Boyle, G S, Firestein
openaire   +2 more sources

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