Results 91 to 100 of about 8,035 (194)

Reduced Th22 cell proportion and prevention of atopic dermatitis in infants following maternal probiotic supplementation [PDF]

open access: yes, 2017
Background In the randomized, controlled study Probiotics in the Prevention of Allergy among Children in Trondheim (ProPACT), maternal probiotic supplementation reduced the incidence of atopic dermatitis (AD) in the offspring.
Simpson, Melanie Rae   +6 more
core   +2 more sources

Supplementary Material for: Staphylococcal Exotoxins Induce Interleukin 22 in Human Th22 Cells

open access: yes, 2017
Background: We have shown previously that T cells from atopic dermatitis (AD) patients produce more IL-22 upon staphylococcal exotoxin stimulation compared to psoriasis patients and healthy controls.
Satzger I. (3355949)   +4 more
core   +1 more source

Circulating Th1, Th2, Th9, Th17, Th22, and Treg Levels in Aortic Dissection Patients

open access: yesMediators of Inflammation, 2018
Background. Previous studies demonstrated that the subsets of CD4+ T helper (Th) cells are closely related to vascular diseases, including atherosclerosis and hypertension.
Jing Ye   +9 more
doaj   +1 more source

Medroxyprogesterone Acetate Decreases Th1, Th17, and Increases Th22 Responses via AHR Signaling Which Could Affect Susceptibility to Infections and Inflammatory Disease

open access: yesFrontiers in Immunology, 2019
A synthetic progestin, medroxyprogesterone acetate (MPA), was used in a novel study to determine progestin effects on human purified macrophages and Th1, Th2, Th17, Th22 cells. MPA concentrations were equivalent to those in the serum of women after 6 and
Marie-Pierre Piccinni   +6 more
doaj   +1 more source

Correlation between the percentages of Th22 cells as well as Th17 cells and CRP levels as well as DAS28.

open access: yes, 2012
Correlation between the percentages of Th22 cells as well as Th17 cells and CRP levels as well as DAS28.
Lei Qi (173882)   +11 more
core   +1 more source

Triple Cytokine FluoroSpot Analysis of Human Antigen-Specific IFN-γ, IL-17A and IL-22 Responses

open access: yesCells, 2014
The involvement of T-helper (Th)1, Th17 and Th22 cell subsets, in immunity, as well as in pathological inflammatory reactions, makes it important to determine their relative proportion. A triple FluoroSpot detecting the hallmark cytokines of Th1 (IFN-γ),
Tomas Dillenbeck   +3 more
doaj   +1 more source

T Helper 22 Pathway Evaluation in Type 1 Diabetes and Its Complications

open access: yesIranian Journal of Allergy, Asthma and Immunology, 2018
A subset of CD4+ T cells named T helper (Th)22 cells play some pathogenic roles in some autoimmune disorders such as type 1 diabetes (T1D). We aimed to study the correlation between the circulatory number of these cells and serum levels of its related ...
Hossein Akbari   +4 more
doaj  

Th22 cells as well as Th17 cells expand differentially in patients with early-stage and late-stage myelodysplastic syndrome.

open access: yesPLoS ONE, 2012
BackgroundImmunological mechanisms are increasingly recognized in the progression of myelodysplastic syndrome (MDS). Early-stage MDS (E-MDS) is characterized by autoimmune-mediated myelosuppression whereas late-stage MDS (L-MDS) involves immune evasion ...
Lin-lin Shao   +11 more
doaj   +1 more source

Downregulation of RUNX3 moderates the frequency of Th17 and Th22 cells in patients with psoriasis

open access: yesMolecular Medicine Reports, 2016
Psoriasis is a common chronic inflammatory and T cell-meditated skin disease. Runt-related transcription factor 3 (RUNX3), one of the runt‑domain family of transcription factors, has been reported to be a susceptibility gene for psoriasis. The present study was designed to delineate the role and underlying mechanism of RUNX3 involved in the ...
FU, DANDAN   +5 more
openaire   +3 more sources

Unbalanced Aryl Hydrocarbon Receptor Expression in Peripheral and Lesional T Cell Subsets of Atopic Dermatitis

open access: yesClinical, Cosmetic and Investigational Dermatology, 2023
Yu-Qing Hu, Jian-Zhong Zhang Department of Dermatology, Peking University People’s Hospital, Beijing, 100044, People’s Republic of ChinaCorrespondence: Jian-Zhong Zhang, Department of Dermatology, Peking University People’s Hospital, No. 11 South Avenue,
Hu YQ, Zhang JZ
doaj  

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