Results 151 to 160 of about 116,173 (199)

Matrix metalloproteinase activation and TNF upregulation characterize the sclerotic phase of aortic valve disease. [PDF]

Br J Biomed Sci
Chan-Delgado E, Lerma C, Echeverría JC, Toledo A, Torres-Arellano JM, Infante O, Bojalil R, Cossío-Aranda JE, Ávila-Vanzzini N, Amezcua-Guerra LM, Springall R.   +10 more
europepmc   +1 more source

Effects of P38 MAPK Pathway Inhibition on the Metabolism of Periodontal Ligament Fibroblasts During Inflammation. [PDF]

J Cell Mol Med
Qian C, Gu Y, Gao F, Xu Z, Li J, Liu S, He R, Zhong L, Wang J.   +8 more
europepmc   +1 more source

Baseline stool TIMP-2 predicts strictures and penetrating disease progression in Crohn's patients. [PDF]

Front Immunol
Polamarasetty H, Pereira R, Maruvada V, Vanarsa K, Wankhade D, Yadavalli R, Kugathasan S, Mohan C.   +7 more
europepmc   +1 more source

Sex Hormone Imbalance in Keratoconus: Estrogen-Mediated Collagen Remodeling and Biomechanical Weakening. [PDF]

Invest Ophthalmol Vis Sci
Zhang M, Liu Y, Gao N, Ma Y, Li F, Li X, Cui M, Zhao S, Yang R.   +8 more
europepmc   +1 more source

Hypoxia regulates epithelial to mesenchymal transition-associated genes in human trophoblast cells by modulating DNA methylation. [PDF]

PLoS One
Choudhury J, Dhole B, Aggarwal K, Singh P, Jala MA, Pandey D, Chaturvedi PK, Gupta S.   +7 more
europepmc   +1 more source

Substrate stiffness modulates human gingival fibroblast paracrine signaling to promote osteogenic differentiation of human periodontal ligament cells. [PDF]

Front Bioeng Biotechnol
Tiskratok W, Yamada M, Kyawsoewin M, Lwin HY, Egusa H, Jitprasertwong P, Limraksasin P.   +6 more
europepmc   +1 more source

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Matrix metalloproteinase‐2 regulates the expression of tissue inhibitor of matrix metalloproteinase‐2

Clinical and Experimental Pharmacology and Physiology, 2010
Summary1. Matrix metalloproteinases (MMP) are associated with the vascular remodelling seen in atherosclerosis and aneurysm. The activation and activity of MMP‐2 are regulated by the intrinsic tissue inhibitor of MMP‐2 (TIMP‐2). The aim of the present study was to examine whether, conversely, MMP‐2 can affect the gene and protein expression of TIMP‐2.2.
Kaoru, Kimura, Xian Wu, Cheng, Kae, Nakamura, Aiko, Inoue, Lina, Hu, Haizhen, Song, Kenji, Okumura, Akihisa, Iguchi, Toyoaki, Murohara, Masafumi, Kuzuya   +9 more
openaire   +2 more sources

Polymorphisms in the matrix metalloproteinase‐2 and tissue inhibitor of metalloproteinase‐2 and the risk of human adenomyosis

Environmental and Molecular Mutagenesis, 2008
AbstractThe matrix metalloproteinases (MMPs) and tissue inhibitor of metalloproteinases (TIMPs) may contribute to the development of adenomyosis. The aim of the present study was to investigate whether three single nucleotide polymorphisms (SNPs) in the promoter regions of MMP‐2 (−1306C/T and −735C/T) and TIMP‐2 (‐418G/C) genes were related to the risk
Shan, Kang, Xiwa, Zhao, Huimin, Xing, Na, Wang, Rongmiao, Zhou, Shucheng, Chen, Wansheng, Li, Jian, Zhao, Yanan, Duan, Donglan, Sun, Yan, Li   +10 more
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Endometrial tumor invasiveness is related to metalloproteinase 2 and tissue inhibitor of metalloproteinase 2 expressions

International Journal of Gynecological Cancer, 2006
Matrix metalloproteinase (MMPs) expression has been linked to gynecological tumor aggressiveness. The objective of this study was to determine MMP-2, MMP-7, and MMP-9 and tissue inhibitors of metalloproteinases (TIMP)-1 and TIMP-2 expression in endometrial malignancies and their relation to clinical and histologic parameters.
Graesslin, Olivier, Cortez, Annie, Uzan, C., Birembaut, Philippe, Quereux, Christian, Daraï, Emile   +5 more
openaire   +4 more sources

Erythropoietin Modulates Imbalance of Matrix Metalloproteinase-2 and Tissue Inhibitor of Metalloproteinase-2 in Doxorubicin-induced Cardiotoxicity

Heart, Lung and Circulation, 2014
Doxorubicin (DOX) is a highly effective anti-cancer drug with limited clinical use due to its serious cardiotoxicity. Recent studies reported that erythropoietin (EPO) could exert a cardioprotective effect by non-erythropoietic effects. This study was to investigate fibrosis of DOX-induced cardiotoxicity and determine mechanisms of EPO against ...
Xing, Chen, Zhihua, Guo, Peixian, Wang, Miqing, Xu   +3 more
openaire   +2 more sources