Results 241 to 250 of about 151,127 (263)
Diverse Effects of Various Toll-Like Receptor 2 Ligands on Neuronal Activity and Cell Death. [PDF]
Cell Mol NeurobiolSato F, Hachimura S.
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Vaccination with Carbapenemase KPC-2 and Virulence Factor Pal Provided Robust Protection Against <i>Klebsiella pneumoniae</i> Lung Infection. [PDF]
Vaccines (Basel)Jiang S +13 more
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Unraveling the Mechanism of Cuochuangling Pills in Rosacea Treatment: An Integrated Approach Combining Network Pharmacology, Bioinformatics, and Experimental Validation. [PDF]
Clin Cosmet Investig DermatolShen X +5 more
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Science Signaling, 2021
TLR2 located in the endothelium promotes angiogenesis and inflammation.
Samantha Mahfoud, Tatiana V. Petrova
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TLR2 located in the endothelium promotes angiogenesis and inflammation.
Samantha Mahfoud, Tatiana V. Petrova
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TLR2 in murine atherosclerosis
Seminars in Immunopathology, 2007Atherosclerosis was once thought to be solely a disease of lipid accumulation in the vessel wall. It does involve lipid accumulation, but inflammation appears to be an important driving factor. Consequently, our laboratory undertook to examine the role(s) of TLRs, and especially TLR2, in murine models of atherosclerosis.
Peter S, Tobias, Linda K, Curtiss
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Immunology, 2023
AbstractLeishmania donovani, a protozoan parasite, resides and replicates in macrophages and inflicts the potentially fatal disease visceral leishmaniasis (VL). The parasite‐expressed surface lipophosphoglycan (LPG) was implicated in binding TLR2 on NK cells, but the modus operandi of its disease‐promoting influence remained unknown.
Soumya Kanti Ghosh +6 more
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AbstractLeishmania donovani, a protozoan parasite, resides and replicates in macrophages and inflicts the potentially fatal disease visceral leishmaniasis (VL). The parasite‐expressed surface lipophosphoglycan (LPG) was implicated in binding TLR2 on NK cells, but the modus operandi of its disease‐promoting influence remained unknown.
Soumya Kanti Ghosh +6 more
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Rare TLR2 mutations reduce TLR2 receptor function and can increase atopy risk
Allergy, 2009Background: Common genetic variations in toll‐like receptor 2 (TLR2), an innate pathogen recognition receptor, may influence the development of atopic diseases. So far, very little is known about the role of rare TLR2 mutations in these diseases.Objective: We investigated the functional properties of six rare amino acid changes in TLR2 (and one amino
M S D, Kormann +9 more
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