Results 111 to 120 of about 3,516,666 (358)
Asian Journal of Tropical Biotechnology, 2004 The caused of disability on cerebral palsy is unknown, but the human cerebral hypoxic, which affected to the motor cortex and motor pathways in the brain is believed cause of it. The major clinical manifestations are abnormality of movements and posture patterns. The immunity against the cerebral palsy (CP) is unknown, but interleukine-1β (IL-1β) and
openaire +1 more source
FEBS Open Bio, EarlyView.In a murine model of myocardial ischemia and reperfusion (MI/R), the CD36 azapeptide ligand MPE‐298 reduces cardiac injury and transiently lowers left ventricular long‐chain fatty acids (LCFAs) accumulation 3 h after reperfusion, accompanied by a decrease of oxidative stress and inflammation‐associated genes' expression in the heart and adipose tissue.
Jade Gauvin +12 more
wiley +1 more source
ヒト歯肉上皮細胞のInterleukin-8産生に対するtumor necrosis factor -αの影響 [PDF]
大阪歯科大学Osaka Dental University博士（歯学）Tumor necrosis factor (TNF)-α is an inflammatory mediator that is released during the early stages of the inflammatory response and plays an important role in the development of periodontitis.
鄭, 峰, Zheng, Feng, テイ, ホウ
core +1 more source
TNF-α expression and age in skin punch biopsies of healthy controls and PD patients.
, 2022 Age is not correlated with altered TNF-α gene expression in the skin punch biopsy samples obtained from the lateral lower leg of healthy controls (a, b) and PD patients (c, d). Abbreviations: PD = Parkinson’s disease, TNF-α = tumor necrosis factor-alpha.
Anastasia Kuzkina (14019707) +5 more
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Jurnal Kedokteran Brawijaya, 2014 Kejang demam adalah penyebab kejang paling umum pada anak dan sering menjadi penyebab rawat inap di rumah sakit secara darurat. Studi pendahuluan pada anak menunjukkan bahwa jaringan sitokin diaktifkan dan mungkin berperan dalam patogenesis kejang demam namun, signifikansi klinis yang tepat masih belum jelas.
Dewi Nurindah +2 more
openaire +2 more sources
FEBS Open Bio, EarlyView.Repair of neuronal DNA damage in Alzheimer's disease by KCL‐286. (A) Amyloid‐β oligomers and plaques impair neuronal DNA repair pathways, leading to DNA double‐strand breaks and glial activation. (B) KCL‐286 activates RARβ/RXR signalling via retinoic acid response elements (RAREs), associated with increased BRCA1 expression, enhanced DNA repair and ...
Natasha Hill +6 more
wiley +1 more source
The inflammatory effects of TNF-α and complement component 3 on coagulation
Scientific Reports, 2018 Tissue necrosis factor-α (TNF-α) and complement component 3 (C3) are two well-known pro-inflammatory molecules. When TNF-α is upregulated, it contributes to changes in coagulation and causes C3 induction.
M. Page, J. Bester, E. Pretorius
semanticscholar +1 more source
MagmaFlow: A desktop platform for artificial intelligence‐driven expression analysis
FEBS Open Bio, EarlyView.MagmaFlow is a free, no‐code platform for gene expression analysis. It generates interactive volcano plots, links genes to literature, pathways, and diseases, prioritizes candidates using millions of publications, identifies affected biological processes, builds network diagrams, and exports publication‐ready figures and reports for macOS and Windows ...
Carlos E. Buss +7 more
wiley +1 more source
TNF-α requires autophagy to induce migration in A7r5 cells.
, 2018 (A) Assessment of migration by the wound healing and transwell assays in A7r5 cells stimulated with TNF-α (100 ng/mL) for 24 h in the presence or absence of chloroquine (CQ, 20 μmol/L) during the last 4 h of TNF-α stimulus (n = 4; **pC) siScramble and ...
Nicole Cancino-Arenas (5210993) +9 more
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Reumatismo, 2002 Objectives: To study –238 and +489 TNF-α polymorphisms in severe-unresponsive (more than 6 swollen joints and still active disease despite at least 6 months of DMARDs combination therapy) and mild-responsive (less than 3 swollen joints and good response ...
M. Fabris +5 more
doaj +1 more source