Results 191 to 200 of about 15,449 (239)
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Ciprofloxacin‐induced toxic optic neuropathy

Clinical & Experimental Ophthalmology, 2007
AbstractA case of bilateral toxic optic neuropathy that developed in the setting of prolonged oral ciprofloxacin use for the treatment of chronic osteomyelitis is described. The patient experienced progressive, bilateral and symmetrical visual loss, central scotomas and loss of colour vision over a 2‐month period.
Nadeeka Samarakoon   +2 more
openaire   +3 more sources

Cuban Epidemic Optic Neuropathy and its Relationship to Toxic and Hereditary Optic Neuropathy

Seminars in Ophthalmology, 2010
The similarities and differences between toxic/nutritional and hereditary optic neuropathy and the pathophysiologic mechanisms that they have in common are described. This is based on data from the epidemic suffered in Cuba in 1992, which affected the optic nerves of many individuals and the experience of the authors in dealing with various toxic optic
Rosaralis, Santiesteban-Freixas   +5 more
openaire   +4 more sources

Drug-induced toxic optic neuropathy

Vestnik oftal'mologii, 2020
Drug-induced optic neuropathy is a group of disorders in which medications cause degeneration of the optic nerve. The true prevalence of drug-induced neuropathy has not been studied, although the percentage of patients who develop optic nerve damage is known for individual medications.
O D, Ostroumova   +5 more
openaire   +2 more sources

Toxic Optic Neuropathy From Quinine Overdose

Journal of Neuro-Ophthalmology, 2020
Abstract: A 45-year-old man presented with longstanding poor vision in both eyes. His medical history was significant for a remote overdose of quinine. After the ingestion, he fell into a coma and on awakening was not able to see light out of both eyes. Several days later, his central vision began to gradually recover and continued to improve
Paul R, Freund   +2 more
openaire   +2 more sources

Linezolid-associated toxic optic neuropathy

Neurology, 2006
The oxazolidinone antimicrobial linezolid is effective against gram-positive bacteria. Although maximal recommended therapy is 28 days, treatment durations greater than this are common. Linezolid may cause reversible optic neuropathy and irreversible peripheral neuropathy after months of treatment.
J C, Rucker   +4 more
openaire   +2 more sources

Toxic optic neuropathies

Current Opinion in Ophthalmology, 2013
Many causes of toxic optic neuropathy have been described to date and novel causes of toxicity are continuously being added to the current literature. The pathophysiological basis for the toxicity or a direct causal relationship is yet to be determined for many of these agents.
openaire   +2 more sources

Toxic optic neuropathies: an updated review

Acta Ophthalmologica, 2014
AbstractToxic optic neuropathy (TON) is caused by the damage to the optic nerve through different toxins, including drugs, metals, organic solvents, methanol and carbon dioxide. A similar clinical picture may also be caused by nutritional deficits, including B vitamins, folic acid and proteins with sulphur‐containing amino acids. This review summarizes
Andrzej, Grzybowski   +3 more
openaire   +2 more sources

Ethambutol/Linezolid Toxic Optic Neuropathy

Optometry and Vision Science, 2016
To report a rare toxic optic neuropathy after long-term use of two medications: ethambutol and linezolid.A 65-year-old man presented to the Miami Veterans Affairs Medical Center in December 2014 for evaluation of progressive vision decrease in both eyes.
openaire   +2 more sources

Toluene Related Toxic Optic Neuropathy

Neuro-Ophthalmology, 2009
ABSTRACTToxic optic neuropathy presenting as bilateral painless visual loss causing central or caecocentral scotoma has a wide differential diagnosis. In this report, we present a rare cause of toxic optic neuropathy presenting as almost indistinct optic disc margins in both eyes in a 30-year old man with blurred vision and difficulty in walking and in
Fatih C. Gundogan   +5 more
openaire   +1 more source

Toxic-Metabolic and Hereditary Optic Neuropathies

Continuum, 2019
The diagnosis of visual loss from toxic-metabolic and hereditary optic neuropathies may be delayed in some cases because of a failure to elicit important information in the clinical history or to recognize typical examination findings. An understanding of the features specific to each type of toxic-metabolic and hereditary optic neuropathy, and of the ...
openaire   +2 more sources

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