Results 71 to 80 of about 8,985 (178)

IL-26 in allergic contact dermatitis: resource in a state of readiness [PDF]

open access: yes, 2018
In this study, we investigated the role of IL-26 in allergic contact dermatitis (ACD), highlighting its contribute in the cytotoxic mechanism responsible of the tissue injury.
Balato   +11 more
core   +1 more source

Low anti-staphylococcal IgG responses in granulomatosis with polyangiitis patients despite long-term Staphylococcus aureus exposure [PDF]

open access: yes, 2015
Chronic nasal carriage of the bacterium Staphylococcus aureus in patients with the autoimmune disease granulomatosis with polyangiitis (GPA) is a risk factor for disease relapse.
Arends, J.P.A. (Jan)   +18 more
core   +1 more source

Staphylococcal Superantigen (TSST-1) Mutant Analysis Reveals that T Cell Activation Is Required for Biological Effects in the Rabbit Including the Cytokine Storm

open access: yesToxins, 2010
Staphylococcal superantigens (sAgs), such as toxic shock syndrome toxin 1 (TSST-1), induce massive cytokine production, which may result in toxic shock syndrome (TSS) and sepsis.
Norbert Stich   +3 more
doaj   +1 more source

Interaction of staphylococcal fibronectin binding protein (FnBP), TSST-1 and α-toxin with murine lymphocytes [PDF]

open access: yesFEMS Microbiology Letters, 1992
The most restrictive factor in the inductive phase of immune response is the contact and adhesion of antigen-presenting cells and responding lymphocytes. We report the in vitro formation of aggregates (clusters) of murine T lymphocytes and dendritic cells (DC) pulsed with highly purified staphylococcal toxins (TSST-1, alpha-toxin) and gene-cloned ...
B, Rozalska, T, Wadström
openaire   +2 more sources

A Modified Trier Social Stress Test for Vulnerable Mexican American Adolescents. [PDF]

open access: yes, 2017
The Trier Social Stress Test (TSST) is a well validated and widely used social stressor that has been shown to induce a 2-4 fold increase in cortisol, the biological output produced by the Hypothalamic-Pituitary-Adrenal (HPA) axis in humans.
Alkon, Abbey   +5 more
core   +2 more sources

The Prevalence of Toxin Shock Syndrome oxin ( TSST-1) Producing Clinical Isolates of Staphylococcus aureus StrainsIsolated from Shohada Hospital in Tabriz, Iran

open access: yesMedical Laboratory Journal, 2011
Background and objectives: Staphylococcus aureus is one of the most important etiological agents of hospital and community acquired infections. The enterotoxins and toxin shock syndrome toxin (TSST-1) are among the most common virulent determinants of ...
Teyhoo M   +5 more
doaj  

Antimicrobial peptides effectively kill a broad spectrum of Listeria monocytogenes and Staphylococcus aureus strains independently of origin, sub-type, or virulence factor expression [PDF]

open access: yes, 2008
Background Host defense peptides (HDPs), or antimicrobial peptides (AMPs), are important components of the innate immune system that bacterial pathogens must overcome to establish an infection and HDPs have been suggested as novel antimicrobial ...
Caroline Gottlieb   +5 more
core   +3 more sources

Expression of toxic shock syndrome toxin-1 gene of Staphylococcus aureus in milk: Proof of concept

open access: yesMljekarstvo, 2018
The aim of this study was to assess the expression of a toxic shock syndrome toxin-1 (TSST-1) gene of Staphylococcus aureus in different types of milk, depending on inadequate temperature and storage conditions.
Milijana Babić   +6 more
doaj   +1 more source

Human Keratinocyte Response to Superantigens

open access: yesmSphere, 2020
Staphylococcus aureus and Streptococcus pyogenes are significant human pathogens, causing infections at multiple body sites, including across the skin. Both are organisms that cause human diseases and secrete superantigens, including toxic shock syndrome
Patrick M. Schlievert   +3 more
doaj   +1 more source

Sirtuin 3 deficiency does not alter host defenses against bacterial and fungal infections. [PDF]

open access: yes, 2017
Sirtuin 3 (SIRT3) is the main mitochondrial deacetylase. SIRT3 regulates cell metabolism and redox homeostasis, and protects from aging and age-associated pathologies. SIRT3 may drive both oncogenic and tumor-suppressive effects.
Acha-Orbea, H.   +6 more
core   +1 more source

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