Results 271 to 280 of about 1,825,715 (311)

The Replication of Bluetongue Virus

1990
Bluetongue virus (BTV) replicates in the cytoplasm of a wide variety of cell types and infection ultimately leads to cell death. The studies of Verwoerd, Huismans and others in the late 1960s and continuing to the present (see Chap. 2, this volume) on the double-stranded, segmented genomic RNA (Verwoerd 1969; Verwoerd et al. 1970), the bishelled nature
B T, Eaton, A D, Hyatt, S M, Brookes
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Replication of the hepatitis C virus

Best Practice & Research Clinical Gastroenterology, 2000
Infection with the hepatitis C virus (HCV) is a major cause of chronic liver disease. HCV is an enveloped plus-strand RNA virus closely related to flavi- and pestiviruses. The first cloning of the HCV genome, about 10 years ago, initiated research efforts leading to the elucidation of the genomic organization and the definition of the functions of most
R, Bartenschlager, V, Lohmann
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The replication of cowpea mosaic virus

BioEssays, 1986
AbstractCowpea mosaic virus (CPMV) is the type member of the comovirus group, which contains 14 different plant viruses that have the same structural organization of genomic RNAs and virions and use the same mechanism for expression of the viral RNAs. The combined structure and organization of the two CPMV genomic RNAs is strikingly similar to that of ...
van Kammen, A., Eggen, R.I.L.
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The Measles Virus Replication Cycle

2008
This review describes the two interrelated and interdependent processes of transcription and replication for measles virus. First, we concentrate on the ribonucleoprotein (RNP) complex, which contains the negative sense genomic template and in encapsidated in every virion.
B K, Rima, W P, Duprex
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Replication of hepatitis C virus

Journal of Viral Hepatitis, 1996
SUMMARY. The mode of replication of the hepatitis C virus (HCV) remains poorly understood. Attempts to produce a tissue culture model containing replicating HCV have been largely unsuccessful. Recent studies on sera from patients chronically infected with HCV have shown that viral particles may be found in high‐or low‐density fractions.
H, Yoshikura, M, Hijikata, N, Nakajima, Y K, Shimizu   +3 more
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Retroid Virus Genome Replication

1987
Publisher Summary Sixteen years have passed since reverse transcription was demonstrated in the replication cycle of retroviruses. This unique observation rationalized many earlier studies demonstrating that inhibitors of DNA synthesis prevented the establishment of infection by these RNA viruses.
W S, Mason, J M, Taylor, R, Hull
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On the structure and replication of influenza virus

Journal of Molecular Biology, 1967
Abstract Influenza virus PR8 has been purified from the allantoic fluid of infected chick embryos and from tissue culture medium. Preliminary characterization indicates that the virus contains at least two major proteins and several RNA components with sedimentation coefficients ( S 20, w ) ranging from about 9 to 18 s. Synthesis of virus-specific
P H, Deusberg, W S, Robinson
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Influenza Virus Transcription and Replication

2013
The influenza A viruses cause yearly epidemics and occasional pandemics of respiratory disease, which constitute a serious health and economic burden. Their genome consists of eight single-stranded, negative-polarity RNAs that associate to the RNA polymerase and many nucleoprotein monomers to form ribonucleoprotein complexes (RNPs).
Jaime, Martín-Benito, Juan, Ortín
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Replication strategies of rabies virus

Virus Research, 2005
Rabies virus (RV) is a prototype neurotropic virus that causes fatal disease in human and animals. RV infects hosts at the periphery, enters motoneurons or sensory nerves and moves to the central nervous system (CNS) via retrograde axonal transport. At later stages, there is also centrifugal spread to major exit portals, such as the salivary glands ...
Stefan, Finke, Karl-Klaus, Conzelmann
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Replication of the influenza virus genome

Virology, 1982
Abstract Investigations of the synthesis of virus RNA (vRNA) in chick cells infected with fowl plague virus have shown that the eight vRNAs are produced in nonequivalent amounts and in different relative proportions at various times during infection.
G L, Smith, A J, Hay
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