Results 31 to 40 of about 66,180 (284)
Alteration in different lipoprotein fractions in chronic renal failure patients
MRIMS Journal of Health Sciences, 2016 Background: Chronic renal failure, which is an inevitable terminal pathway of any chronic renal parenchymal disease, is more known for its morbidity than for its mortality.
M Ravi Kumar, Ravindra Kumar
doaj +1 more source
Journal of Clinical Endocrinology and Metabolism, 2016 CONTEXT Randomized controlled trials in nonalcoholic fatty liver disease (NAFLD) have shown that regular exercise, even without calorie restriction, reduces liver steatosis.
F. Shojaee-Moradie +9 more
semanticscholar +1 more source
VLDL Selection into VLDL Transport Vesicle (VTV) is Regulated by CideB
The FASEB Journal, 2012 Very low‐density lipoproteins (VLDLs) circulating in the plasma play a key role in the development of atherosclerosis. VLDLs are synthesized and secreted by the liver. The rate‐determining step in VLDL secretion is their transport from the endoplasmic reticulum (ER) to the Golgi, which is facilitated by a distinct vesicle, the VTV.
Samata Tiwari +3 more
openaire +1 more source
VLDL Triglyceride Kinetics in Lean, Overweight, and Obese Men and Women.
Journal of Clinical Endocrinology and Metabolism, 2016 CONTEXT High-plasma very low-density lipoprotein (VLDL) triglyceride (TG) concentration and alterations in VLDL-TG metabolism are associated with cardiometabolic disease.
B. Mittendorfer +3 more
semanticscholar +1 more source
Journal of atherosclerosis and thrombosis, 2017 Very low-density lipoprotein (VLDL) receptor is a member of the low-density lipoprotein (LDL) receptor family. It binds triglyceride rich lipoprotein (TGRL) but not LDL, because it recognizes apolipoprotein (apo)E only but not apoB.
Sadao Takahashi
semanticscholar +1 more source
Gene expression and intracellular pathways involved in endothelial dysfunction induced by VLDL and oxidised VLDL [PDF]
Cardiovascular Research, 2003 The molecular mechanisms underlying the relationship between elevated plasma concentrations of triglyceride-rich lipoproteins and coronary artery disease remain uncertain. In the present work, we investigated the gene expression pattern and intracellular pathways in human endothelial cells incubated with very low density lipoproteins (VLDL).
G.D. Norata +5 more
openaire +4 more sources
Inactivation of ANGPTL3 reduces hepatic VLDL-triglyceride secretion1[S]
Journal of Lipid Research, 2015 Humans and mice lacking angiopoietin-like protein 3 (ANGPTL3) have pan-hypolipidemia. ANGPTL3 inhibits two intravascular lipases, LPL and endothelial lipase, and the low plasma TG and HDL-cholesterol levels in ANGPTL3 deficiency reflect increased ...
Yan Wang +5 more
semanticscholar +1 more source
Acta Biomedica Scientifica, 2016 Hyperglycemia and dyslipidemia are common metabolic abnormalities in adults with type 1 diabetes mellitus (T1DM) and both increase cardiovascular disease risk.
V. S. Syomshchikov +2 more
doaj +1 more source
Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics, 1977 血中リポ蛋白の中間代謝産物 (ILDL, remnant) が異常に増加するIII型高脂血症は, 血清 Chol, TG値からは診断できず, 超遠心分離および電気泳動により, β-VLDLを証明する必要がある. しかしβ-VLDLの証明はやや客観性に乏しく, 本症診断における特異性にも問題がある. 最近III型高脂血症に特異的で感度の高い診断方法が数多く報告されているが, 未だ十分なものはない. 今回我々は, 臨床像およびリポ蛋白泳動によりβ-VLDLを証明した典型的なIII型高脂血症と思われる症例の血清脂質およびリポ蛋白を頻回に測定し, 他のタイプの高脂血症と比較検討し, 以下の結論を得た.1) III型高脂血症の血清 Chol は182～540mg/dlでTGは142～898mg/dlであった.
Hiroshi Mabuchi +10 more
openaire +4 more sources
Current Issues in Pharmacy and Medical Sciences, 2017 The aim of this paper was to examine whether moderate dyslipoproteinemia can cause an increase of hsCRP and LPO levels in Tx patients who had received immunosuppressive therapy and were without acute inflammatory diseases. Herein, the lipid levels, hsCRP,
Kimak Elzbieta +2 more
doaj +1 more source