Cancer Research in Adenocarcinoma, Adenoma, Adenomatous Polyposis Coli, and Colitis-Associated Neoplasia: A Special Issue. [PDF]
Moriichi K, Fujiya M.
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Numerous experimental, epidemiologic, and clinical studies suggest that nonsteroidal anti-inflammatory drugs (NSAIDs), particularly the highly selective cyclooxygenase (COX)-2 inhibitors, have promise as anticancer agents. NSAIDs restore normal apoptosis
Patrono, Carlo
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A pan-cancer analysis on the carcinogenic effect of human adenomatous polyposis coli. [PDF]
Zhang Y, Liu X, Li A, Tang X.
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Association of supernumerary teeth and familial adenomatous polyposis: a case report
Familial adenomatous polyposis (FAP) is an autosomal, dominant disorder caused by mutation of a particular gene, adenomatous polyposis coli (APC). This paper reports a case of a patient with FAP who presented with recurrent formation of supernumerary ...
Anup J Karki +3 more
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To Identify Adenomatous Polyposis Coli Gene Mutation as a Predictive Marker of Endometrial Cancer Immunotherapy. [PDF]
Song Y, Huang J, Wang K, Li Y.
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Familial adenomatous polyposis syndrome (FAP) is a rare multisystemic disorder that affects individuals who have mutation in the adenomatous polyposis coli (APC) gene.
Kenneth A. Agu +6 more
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E74‑like ETS transcription factor 5 facilitates cell proliferation through regulating the expression of adenomatous polyposis coli 2 in non‑small cell lung cancer. [PDF]
Wen J +8 more
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Multivalent Interaction of Beta-Catenin With its Intrinsically Disordered Binding Partner Adenomatous Polyposis Coli. [PDF]
Rowling PJE +3 more
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The in vivo rate of somatic adenomatous polyposis coli mutation.
Cancer arises through successive somatic mutations/epimutations of oncogenes and tumor-suppressor genes. Accurate estimates of the rates at which these (epi)mutations occur are a vital but missing link in our emerging quantitative understanding of ...
Page, KM +3 more
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