Results 31 to 40 of about 5,991,558 (223)

Therapy of Inflammatory Bowel Disease using 5-Aminosalicylic Acid, 4-Aminosalicylic Acid and Olsalazine Retention Enemas: Review of Clinical Trials

open access: yesCanadian Journal of Gastroenterology, 1989
The knowledge that 5-aminosalicylic acid (5-ASA) is the active compound of sulphasalazine and that it acts topically has stimulated the use of 5-ASA containing enemas and ocher similar compounds such as olsalazine and 4-aminosalicylic acid (4-ASA). 5-ASA
M. Campieri   +6 more
doaj   +1 more source

Nephrotic syndrome after treatment of Crohn′s disease with mesalamine: Case report and literature review

open access: yesAvicenna Journal of Medicine, 2012
Inflammatory bowel disease and its various treatments may affect the kidney in several ways. Recently, case reports have been published documenting the development of nephrotic syndrome after the treatment for inflammatory bowel disease with 5 ...
Belal M Firwana   +6 more
doaj   +1 more source

Crystal structure of methyl 2-hydroxy-5-[(4-oxo-4,5-dihydro-1,3-thiazol-2-yl)amino]benzoate

open access: yesActa Crystallographica Section E: Crystallographic Communications, 2015
The title compound, C11H10N2O4S, crystallized with two independent molecules (A and B) in the asymmetric unit. They differ primarily in the rotational orientation of the five-membered heterocyclic ring.
Shaaban K. Mohamed   +4 more
doaj   +1 more source

5-Aminosalicylic Acid-induced Myocarditis in a Patient with Atypical Ulcerative Colitis

open access: yesThe Korean Journal of Gastroenterology, 2022
5-aminosalicylic acid (5-ASA) is used widely to treat ulcerative colitis. The common side effects of 5-ASA include nausea, vomiting, abdominal pain, headache, and skin rash.
Hyo Yeop Song, Geom Seog Seo
doaj   +1 more source

5-Aminosalicylic Acid Alters the Gut Bacterial Microbiota in Patients With Ulcerative Colitis

open access: yesFrontiers in Microbiology, 2018
Background: The aim of this study was to clarify the effect of 5-aminosalicylic acid (5-ASA) treatment on gut bacterial microbiota in patients with ulcerative colitis (UC).
Jun Xu   +8 more
semanticscholar   +1 more source

Synthesis, crystal structure and Hirshfeld surface analysis of a 1:1 salt of sparfloxacin and 4-aminosalicylic acid

open access: yesActa Crystallographica Section E: Crystallographic Communications
The anhydrous salt of sparfloxacin [5-amino-1-cyclopropyl-7-(3,5-dimethylpiperazin-1-yl)-6,8-difluoro-4-oxo-1,4-dihydroquinoline-3-carboxylic acid] with 4-aminosalicylic acid, C22H22F2N4O3+·C7H6NO3−, features both intermolecular (N—H...O) and ...
Bhumi C. Patel   +2 more
doaj   +1 more source

Toxicity of Gold Nanoparticles in Mice due to Nanoparticle/Drug Interaction Induces Acute Kidney Damage

open access: yesNanoscale Research Letters, 2020
Nanomaterials are innovative materials with many useful properties, but there is concern regarding their many unknown effects on living organisms. Gold nanoparticles are widely used as industrial materials because of their excellent properties.
Katsuhiro Isoda   +9 more
doaj   +1 more source

The Estimation of Para-Aminosalicylic Acid

open access: yesJournal of Pharmacy and Pharmacology, 1954
Summary A comparative study of the existing methods of estimation of p-aminosalicylic acid and its compounds has been made and some of their defects pointed out. A method based on both decarboxylation and diazotisation is proposed and its advantages over the existing methods pointed out by a comparative study.
G, KAMESWARA RAO   +3 more
openaire   +2 more sources

Mucosal 5‐aminosalicylic acid concentration, drug formulation and mucosal microbiome in patients with quiescent ulcerative colitis

open access: yesAlimentary Pharmacology and Therapeutics, 2019
5‐aminosalicylic acid (5‐ASA) is the first‐line therapy for ulcerative colitis (UC). 5‐ASA acts locally in the colonic mucosa by numerous proposed mechanisms, and is metabolised by N‐acetyltransferase (NAT).
M. Olaisen   +10 more
semanticscholar   +1 more source

IL‐7R‐Enriched Extracellular Vesicles From the Thymus Drive Colitis via Promoting Neutrophil Extracellular Trap Formation

open access: yesAdvanced Science, EarlyView.
Inflammatory bowel disease (IBD) pathogenesis remains unclear. This study reveals that thymus‐originated IL‐7R‐enriched extracellular vesicles exacerbate colitis by promoting neutrophil extracellular traps via a thymus‐gut axis, uncovering an inter‐organ communication mechanism in inflammatory bowel disease pathogenesis.
Yao Liao   +21 more
wiley   +1 more source

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