Results 171 to 180 of about 186,580 (246)

Retinoic Acid Informs the Positional Identity of Frontonasal Neural Crest Cells Through Alx Family of Transcription Factors

open access: yesThe FASEB Journal, Volume 40, Issue 12, 30 June 2026.
Retinoic acid (RA) regulates the positional identity of cranial neural crest cells (CNCCs) through the transcription factors Alx1 and Alx4. Upon receiving RA from the local environment, liganded RAR/RXR heterodimers activate the expression of Alx1 and Alx4, which promote the frontonasal identity of CNCCs and inhibit the first pharyngeal arch identity ...
Shuxuan Wu   +7 more
wiley   +1 more source

Single-nucleus ATAC-seq analysis resolves chromatin and transcriptional features of fibrolamellar carcinoma. [PDF]

open access: yesSci Rep
Farghli AR   +13 more
europepmc   +1 more source

Genome‐wide association study in 21,271 individuals identifies 9 novel loci associated with circulating CD34+ hematopoietic stem and progenitor cell levels

open access: yes
HemaSphere, Volume 10, Issue 7, July 2026.
Yuwen Xie   +20 more
wiley   +1 more source

ID3 deficiency alters chromatin accessibility at DSB sites and enhances vulnerability to HDAC inhibition

open access: yesInternational Journal of Cancer, Volume 158, Issue 12, Page 3173-3186, 15 June 2026.
What's new? Errors in DNA double‐strand break (DSB) repair can lead to mutations, chromosomal instability, and ultimately cancer. Inhibitor of DNA‐binding 3 (ID3), a transcriptional repressor, is crucial to promoting DSB repair and helping maintain genome stability. Here, the authors investigated ID3 regulation of DNA repair via chromatin accessibility
Giuditta Della Corte   +10 more
wiley   +1 more source

The Relevance and Resilience of Evo‐Devo in 2025: The Biennial Meeting of the Pan American Society for Evolutionary Developmental Biology

open access: yes
Journal of Experimental Zoology Part B: Molecular and Developmental Evolution, Volume 346, Issue 5, Page 378-382, July 2026.
Mark Rebeiz   +9 more
wiley   +1 more source

Enhanced IFNy response in dedifferentiated melanoma cells is due to chromatin remodeling as revealed by ATAC-seq. [PDF]

open access: yesCell Commun Signal
Sævarsson T   +5 more
europepmc   +1 more source

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