Results 41 to 50 of about 1,179,204 (384)
Neutrinoless $ββ$-decay nuclear matrix elements from two-neutrino $ββ$-decay data [PDF]
We study two-neutrino ($2\nu\beta\beta$) and neutrinoless double-$\beta$ ($0\nu\beta\beta$) decays in the nuclear shell model and proton-neutron quasiparticle random-phase approximation (pnQRPA) frameworks. Calculating the decay half-life of several dozens of nuclei ranging from calcium to xenon with the shell model, and of $\beta\beta$ emitters with a
arxiv +1 more source
Beta-catenin cleavage enhances transcriptional activation [PDF]
AbstractNuclear activation of Wnt/β-catenin signaling is required for cell proliferation in inflammation and cancer. Studies from our group indicate that β-catenin activation in colitis and colorectal cancer (CRC) correlates with increased nuclear levels of β-catenin phosphorylated at serine 552 (pβ-Cat552).
Tatiana Goretsky+12 more
openaire +3 more sources
Expression of dickkopf-1 and beta-catenin related to the prognosis of breast cancer patients with triple negative phenotype. [PDF]
BACKGROUND AND AIM: We investigated the prognostic importance of dickkopf-1(DKK1) and beta-catenin expression in triple negative breast cancers. METHODS: The expression of DKK1 and beta-catenin was evaluated in breast cell lines using RT-PCR and western ...
Wen-Huan Xu+4 more
doaj +1 more source
OSTM1 regulates beta-catenin/Lef1 interaction and is required for Wnt/beta-catenin signaling [PDF]
The Wnt/beta-catenin signaling pathway controls key aspects of embryonic development and adult tissue homeostasis, including the formation and maintenance of bone.
Feigin, M. E., Malbon, C. C.
core +1 more source
A Reaction-Diffusion Model of the Cadherin-Catenin System: A Possible Mechanism for Contact Inhibition and Implications for Tumorigenesis [PDF]
Contact inhibition is the process by which cells switch from a motile growing state to a passive and stabilized state upon touching their neighbors. When two cells touch, an adhesion link is created between them by means of transmembrane E-cadherin proteins.
arxiv +1 more source
Subshifts of finite type and matching for intermediate $β$-transformations [PDF]
We focus on the relationships between matching and subshift of finite type for intermediate $\beta$-transformations $T_{\beta,\alpha}(x)=\beta x+\alpha $ ($\bmod$ 1), where $x\in[0,1]$ and $(\beta,\alpha) \in \Delta:= \{ (\beta, \alpha) \in \mathbb{R}^{2}:\beta \in (1, 2) \; \rm{and} \; 0 < \alpha <2 - \beta\}$.
arxiv +1 more source
In both colon cancer and type 2 diabetes, metabolic changes induced by upregulation of the Wnt/beta-catenin signaling and downregulation of peroxisome proliferator-activated receptor gamma (PPAR gamma) may help account for the frequent association of ...
Y. Lecarpentier+3 more
semanticscholar +1 more source
$γγ$ decay as a probe of neutrinoless $ββ$ decay nuclear matrix elements [PDF]
We study double gamma ($\gamma\gamma$) decay nuclear matrix elements (NMEs) for a wide range of nuclei from titanium to xenon, and explore their relation to neutrinoless double-beta ($0\nu\beta\beta$) NMEs. To favor the comparison, we focus on double-magnetic dipole transitions in the final $\beta\beta$ nuclei, in particular the $\gamma\gamma$ decay of
arxiv +1 more source
The underlying mechanism of Fusobacterium nucleatum (Fn) in the carcinogenesis of colorectal cancer (CRC) is poorly understood. Here, we examined Fn abundance in CRC tissues, as well as β-catenin, TLR4 and PAK1 protein abundance in Fn positive and Fn ...
Yongyu Chen+8 more
semanticscholar +1 more source
Beta-catenin (i.e., canonical Wnt) signaling controls CNS angiogenesis and the blood-brain and blood-retina barriers. To explore the role of the Discs large/membrane-associated guanylate kinase (Dlg/MAGUK) family of scaffolding proteins in beta-catenin ...
Chris Cho+4 more
doaj +1 more source