Results 191 to 200 of about 26,472 (244)
This review systematically summarizes research progress on bismuth‐based semiconductor photocatalytic materials in the field of CO2 reduction. It focuses on elucidating the mechanisms by which four modification strategies—including heteroatom doping, oxygen vacancy engineering, microstructural control, and heterostructure fabrication—regulate band ...
Shuisen He +6 more
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Photothermal CO2 Hydrogenation: Reactions, Mechanisms, and Catalyst Design
Reactions, mechanisms, and catalyst design for photothermal CO2 hydrogenation. ABSTRACT Photothermal CO2 hydrogenation has attracted extensive attention because it provides a green and sustainable approach to fuel and chemicals production. The photothermal catalyst plays a critical role in CO2 conversion through a range of reactions.
Shuai Yan +15 more
wiley +1 more source
ABSTRACT Silicon anodes, with a theoretical capacity of ~ $\unicode{x0007E}$3860 mAh g−1, offer more than ten times the lithium storage of graphite but suffer from large volume expansion during lithiation, leading to particle fracture, electrical disconnection, and unstable solid–electrolyte interphases (SEIs).
Yangyul Ju +6 more
wiley +1 more source
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Autoantibody-Mediated Acquired Deficiency of C1 Inhibitor
New England Journal of Medicine, 1987During the past 25 years, three forms of deficiency of the inhibitor of the first component of complement (C1 inhibitor) with angioedema have been recognized; two forms are hereditary and one is acquired. As compared with hereditary angioedema, the syndrome of acquired C1-inhibitor deficiency is rare, and it is usually associated with ...
Konrad Börk +2 more
exaly +3 more sources
Acquired C1 Esterase Inhibitor Deficiency
Annals of Internal Medicine, 2000Acquired C1 esterase inhibitor deficiency is a rare condition associated with autoimmune or low-grade lymphoproliferative disorders. Adults or elderly patients are most commonly affected. The diagnosis is suspected when patients present with recurrent angioedema and low serum levels of C4 with normal levels of C3.
T, Caballero +2 more
+9 more sources
Best Practice & Research Clinical Gastroenterology, 2005
Hereditary and acquired deficiencies of the C1 inhibitor result in a single prominent symptom, namely angioedema. Angioedema may involve the skin, the gastrointestinal tract or the upper airway. Genetically determined defects in C1INH cause hereditary angioedema.
Fred S, Rosen, Alvin E, Davis
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Hereditary and acquired deficiencies of the C1 inhibitor result in a single prominent symptom, namely angioedema. Angioedema may involve the skin, the gastrointestinal tract or the upper airway. Genetically determined defects in C1INH cause hereditary angioedema.
Fred S, Rosen, Alvin E, Davis
openaire +2 more sources
C1 inhibitor deficiency: management
Clinical and Experimental Dermatology, 2005This is the second of two articles on C1 inhibitor deficiency based on a recent UK consensus document covering its diagnosis and management in adults and children. This summary focuses on the management of the disorder including prophylaxis, emergency treatment and special situations such as pregnancy and dental care.
M M, Gompels, R J, Lock
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C1-inhibitor deficiency and angioedema
Molecular Immunology, 2001C1-inhibitor deficiency can be inherited or acquired; both conditions lead to recurrent angioedema that can be life threatening when the larynx is involved (hereditary angioedema, HAE; acquired angioedema, AAE). The genetic defect is due to the heterozygous deficiency of C1-Inh that is transmitted as an autosomal dominant trait.
A. Carugati +3 more
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Acquired C1 Inhibitor Deficiency
Immunology and Allergy Clinics of North America, 2017Acquired angioedema due to C1-INH deficiency (C1-INH-AAE) can occur when there are acquired (not inherited) deficiencies of C1-INH. A quantitative or functional C1-INH deficiency with negative family history and low C1q is diagnostic of C1-INH-AAE. The most common conditions associated with C1-INH-AAE are autoimmunity and B-cell lymphoproliferative ...
Iris M, Otani, Aleena, Banerji
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Monocyte C1‐inhibitor synthesis in patients with C1‐inhibitor deficiency
European Journal of Clinical Investigation, 1989Abstract. Monocytes of seven out of eight patients with type 1 C1‐inhibitor (C1‐inh) deficiency (HAE) produced 40% as much C1‐inh as monocytes from normal donors (controls). In contrast, monocytes from three patients with type 2 and three patients with acquired C1‐inh deficiency produced similar amounts of Cl‐inh as controls. Recombinant γ‐interferon (
D F, Lappin +6 more
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