Results 221 to 230 of about 38,034 (262)
FURIN Stimulates NOTCH2 and NOTCH3 Pathways, Leading to Return of Function in Aged Cells. [PDF]
Elkin PL +4 more
europepmc +1 more source
Source-Specific Photobiomodulation Regulates Mitochondrial Bioenergetics, Redox Signaling, and Functional Outputs in C2C12 Myoblasts Across Replicative Aging. [PDF]
Aviña AE +8 more
europepmc +1 more source
Skipjack tuna bone derived biocalcium ameliorates C2C12 myotube atrophy through microRNA29b regulation. [PDF]
Jantarawong S +8 more
europepmc +1 more source
Yap is a novel regulator of C2C12 myogenesis
The expression, regulation and function of mammalian Hippo pathway members in skeletal muscle is largely unknown. The aim of this study was thus to test the hypothesis that core members of the mammalian Hippo pathway are expressed in skeletal muscle and that the transcriptional co-factor Yap, a core member of the Hippo pathway, regulates C2C12 ...
Kevin I Watt +2 more
exaly +4 more sources
C2C12 cells: biophysical, biochemical, and immunocytochemical properties
We examined the myofibril biochemical, structural, and biophysical properties of C2C12, a mouse skeletal muscle cell line (American Type Culture Collection), to assess whether force development and the sensitivity of the myofilaments to calcium could be measured in C2C12 myotubes and whether a cardiac contractile protein, troponin T, is expressed and ...
D K, McMahon +6 more
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Effects of hyperoxia and hypoxia on the proliferation of C2C12 myoblasts
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2021Hyperoxic conditions are known to accelerate skeletal muscle regeneration after injuries. In the early phase of regeneration, macrophages invade the injured area and subsequently secrete various growth factors, which regulate myoblast proliferation and differentiation. Although hyperoxic conditions accelerate muscle regeneration, it is unknown whether
Misa, Horiike +2 more
openaire +2 more sources
Prostaglandin D2 inhibits C2C12 myogenesis
Molecular and Cellular Endocrinology, 2010Muscle repair following injury is preceded by a rapid inflammatory response with myoblasts being exposed to high levels of prostaglandin D(2) (PGD(2)) from invading leukocytes. We demonstrate that PGD(2) strongly inhibits C2C12 myogenesis as measured by cell fusion, creatine kinase activity and MyoD, myogenin and alpha-actin expression.
Pedro, Veliça +2 more
openaire +2 more sources

