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Eculizumab in post-transplant C3 glomerulonephritis caused by a C3 mutation .
Clinical Nephrology, 2019C3 glomerulonephritis (C3GN) is a disorder of excess alternative complement activation leading to glomerular injury. Following kidney transplantation, C3GN has a high recurrence rate, and the overall prognosis is poor without treatment.
J. Kim, K. Foster, S. Westphal
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Mini review: A unique case of crescentic C3 glomerulonephritis
Nephrology, 2017 Kidney involvement is an under-recognized complication of non-Hodgkin lymphomas. They occur in a variety of mechanisms and differ widely in their clinical presentation. We take this opportunity to report a case of a 65year-old man who developed a rapidly
D. Palamuthusingam +4 more
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C3 Glomerulonephritis Triggered by Toxocariasis
Journal of the American Society of Nephrology, 2022M. M'rad +4 more
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C3 glomerulonephritis with a severe crescentic phenotype
Pediatric Nephrology, 2017C3 glomerulopathy (C3G) is rare type of glomerulonephritis resulting from the glomerular deposition of C3 due to dysregulation of the alternative pathway of complement. It is further subdivided into C3 glomerulonephritis (C3GN) and dense deposit disease (DDD), depending on the ultrastructural features. C3GN usually presents with a membranoproliferative
A. Ravindran +3 more
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Lupus, 2021
Introduction Activation of the complement pathway by immune complexes is a key feature of systemic lupus erythematosus (SLE) and SLE glomerulonephritis, which translates into low levels of C3 and C4 during active disease.
R. Fernandez-Ruiz +3 more
semanticscholar +1 more source
Introduction Activation of the complement pathway by immune complexes is a key feature of systemic lupus erythematosus (SLE) and SLE glomerulonephritis, which translates into low levels of C3 and C4 during active disease.
R. Fernandez-Ruiz +3 more
semanticscholar +1 more source
De novo C3 glomerulonephritis in a renal allograft
Ultrastructural Pathology, 2016 C3 glomerulonephritis (C3GN) is a recently described, rare glomerular disease characterized by predominant or sole glomerular C3 deposits. Morphologic features of C3GN are similar to those of dense deposit disease (DDD); however, ribbon-like ...
J. Nahm +6 more
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A novel CFHR5 mutation associated with C3 glomerulonephritis in a Turkish girl
JN. Journal of Nephrology (Milano. 1992), 2014 C3 glomerulopathy defines a subgroup of membranoproliferative glomerulonephritis (MPGN) characterized by complement 3 (C3)-positive, immunoglobulin-negative deposits in immunofluorescence microscopy.
N. Beşbaş +4 more
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Membranoproliferative Glomerulonephritis und C3‑Glomerulopathie
Der Internist, 2019Based on an increasingly better pathophysiological understanding over the last 10 years, in 2010 a new classification of glomerulonephritis with dominant or codominant C3 deposits was introduced and the predominant subgoup was termed C3 glomerulopathy (C3G). In the current classification, immune complex mediated membranoproliferative glomerulonephritis
B, Hohenstein, K, Amann, J, Menne
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Nephrology, Dialysis and Transplantation, 2019
BACKGROUND Complement factor H-related protein 5 (CFHR5) nephropathy is an inherited renal disease characterized by microscopic and synpharyngitic macroscopic haematuria, C3 glomerulonephritis and renal failure. It is caused by an internal duplication of
Eleni Frangou +16 more
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BACKGROUND Complement factor H-related protein 5 (CFHR5) nephropathy is an inherited renal disease characterized by microscopic and synpharyngitic macroscopic haematuria, C3 glomerulonephritis and renal failure. It is caused by an internal duplication of
Eleni Frangou +16 more
semanticscholar +1 more source
A case of C3 glomerulonephritis successfully treated with eculizumab
Pediatric Nephrology, 2015C3 glomerulonephritis (C3GN) is a rare form of glomerulopathy that is characterized by predominant C3 deposits. Eculizumab, a humanized monoclonal C5 antibody, has recently emerged as a treatment option for C3GN. We report a C3GN patient successfully treated with eculizumab.A 5-year-old boy who presented with proteinuria, hematuria, high ASO titers ...
Alex Payette +5 more
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