Results 81 to 90 of about 7,336 (225)

212 Suppressor of cytokine signalling 3 (socs3) interaction with cavin-1 links socs3 function and cavin-1 stability [PDF]

Heart, 2017
Caveolae are lipid raft microdomains essential for the compartmentalisation and regulation of several signalling pathways e.g. JAK/STAT signalling. Disruption of caveolae is a significant factor in multiple disorders including muscular dystrophy, cardiovascular disease, and cancer.
Jamie Williams, Nasser Alotaiq, Tim Palmer   +2 more
openaire   +1 more source

Loss of PTRF/cavin-1 increases transmigration. [PDF]

, 2012
Transmigration toward the indicated serum concentration in the lower chamber was measured for (A, n = 4 ...
John F. Hancock (144206), Cho Sanda Aung (144184), Samantha Murphy (144193), Rachael Barry (144199), Fiona Simpson (144202), Sally Martin (144190), Libin Liu (144204), Michelle M. Hill (144181), Debra M. Black (144196), Paul F. Pilch (144205), Dorothy Loo (144187), Noor Huda Daud (144182), Marie-Odile Parat (144207), Robert G. Parton (53)   +13 more
core   +1 more source

Business Papers (MS 80-0003) [PDF]

, 1911
Letter from the Texas Bank and Trust Co.
Texas Bank and Trust Co.
core  

MURC/CAVIN-4 facilitates store-operated calcium entry in neonatal cardiomyocytes.

Biochimica et biophysica acta. Molecular cell research, 2019
Intact store-operated calcium entry (SOCE) mechanisms ensure the maintenance of Ca2+ homeostasis in cardiomyocytes while their dysregulation promotes the development of cardiomyopathies.
Julien Malette, Jade Degrandmaison, Hugo Giguère, Jonathan Berthiaume, M. Frappier, J. Parent, M. Auger-Messier, G. Boulay   +7 more
semanticscholar   +1 more source

Tiny carriers, big impact: How Fusobacterium nucleatum extracellular vesicles drive oral diseases and beyond

Interdisciplinary Medicine, Volume 4, Issue 4, July 2026.
This review explores how Fusobacterium nucleatum extracellular vesicles drive local oral diseases and distal pathologies via sophisticated cross‐compartment cross talk. It highlights the transformative potential of these “tiny carriers” as next‐generation molecular intermediaries for advanced health monitoring and innovative bio‐interventions by ...
Rongyang Ma, Jiahui Huang, Yitong Chen, Haoxuan Wang, Hang Zou, Wanghong Zhao, Zilong Deng   +6 more
wiley   +1 more source

The Effects of Cavin-2 on Angiogenesis [PDF]

, 2016
Caveolae are bulb-shaped invaginations about 50-60nm in size on the plasma membrane of mammalian cells that play a key role in cell signaling, endocytosis, plasma membrane adaptations, and lipid homeostasis.
Pogue, Connor Blaine
core  

Inter-dependent Polarization of caveolin-1 and PTRF/cavin-1. [PDF]

, 2012
Wild type (WT), caveolin-1−/− and PTRF/cavin-1−/− mouse embryo fibroblasts (MEF) were plated on coverslips at low density. Immunostaining was performed with (A) mouse anti-caveolin-1 and rabbit anti-PTRF, (B) rabbit anti-PTRF/cavin-1, or (C) rabbit-anti ...
John F. Hancock (144206), Cho Sanda Aung (144184), Samantha Murphy (144193), Rachael Barry (144199), Fiona Simpson (144202), Sally Martin (144190), Libin Liu (144204), Michelle M. Hill (144181), Debra M. Black (144196), Paul F. Pilch (144205), Dorothy Loo (144187), Noor Huda Daud (144182), Marie-Odile Parat (144207), Robert G. Parton (53)   +13 more
core   +1 more source

Cavin-1 regulates caveolae-mediated LDL transcytosis: crosstalk in an AMPK/eNOS/ NF-κB/Sp1 loop

OncoTarget, 2017
Caveolae are specialized lipid rafts structure in the cell membrane and critical for regulating endothelial functions, e.g. transcytosis of macromolecules like low density lipoprotein (LDL) etc.
X. Bai, Xiao-Yan Yang, Juyi Li, Ye-li, Xiong Jia, Z. Xiong, Yumei Wang, Si Jin   +7 more
semanticscholar   +1 more source

Cavin-1 regulates BMP/Smad signaling through the interaction of Caveolin-1 with BMPRII in pulmonary artery endothelial cells

European Heart Journal, 2020
Pulmonary hypertension (PH) is a progressive disease associated with poor outcome. Caveolin-1 (Cav1) is a component of caveolae and classified as a related gene of pulmonary arterial hypertension (PAH).
S. Tomita, N. Nakanishi, T. Ogata, Y. Tsuji, A. Sakamoto, Y. Higuchi, S. Matoba   +6 more
semanticscholar   +1 more source

NUP85 Mediates Endoplasmic Reticulum Stress through the USP47/ASK1 Signaling Pathway to Regulate the Progression of Liver Fibrosis

Advanced Science, Volume 13, Issue 33, 15 June 2026.
In the present study, we have demonstrated that the NUP85‐USP47‐ASK1 signaling pathway may have regulated the progression of liver fibrosis through modulating ERS. Additionally, we have developed a CREKA‐coupled liposome to target delivery of MV, a pharmacological inhibitor of NUP85, to activated HSCs, thereby attenuating liver fibrosis. ABSTRACT Liver
Dashuai Yang, Haoran Yang, Linxin Pan, Chang Tian, Fucheng Zuo, Shilei Huang, Xianrui Li, Ming Chen, Cheng Qian, Jie Wang, Zhaolin Chen, Tao Xu   +11 more
wiley   +1 more source