Results 231 to 240 of about 69,053 (332)

Over‐expression of Aurora‐A targets cytoplasmic polyadenylation element binding protein and promotes mRNA polyadenylation of Cdk1 and cyclin B1 [PDF]

open access: bronze, 2005
Takashi Sasayama   +7 more
openalex   +1 more source

Cell division responsive peptides for optimized plasmid DNA delivery : the mitotic window of opportunity? [PDF]

open access: yes, 2014
De Smedt, Stefaan   +4 more
core   +1 more source

ZNF224 enhances the oncogenic function of p21 via p53 and AKT pathways in melanoma

open access: yesThe FEBS Journal, EarlyView.
KRAB zinc finger protein ZNF224 acts as an oncogenic factor in melanoma cells, enhancing p21 cytosolic retention and impairing nuclear p21 cytostatic activity through stimulation of the AKT pathway. Furthermore, ZNF224 regulates transcriptional expression levels of p21 via p53.
Leandra Sepe   +12 more
wiley   +1 more source

BUB1 and CENP-U, Primed by CDK1, Are the Main PLK1 Kinetochore Receptors in Mitosis

open access: yesMolecules and Cells, 2020
Priyanka Singh   +8 more
semanticscholar   +1 more source

Neocortical neurogenesis: a proneural gene perspective

open access: yesThe FEBS Journal, EarlyView.
The neocortex is a mammalian‐specific brain region responsible for higher‐order cognitive functioning that shares fundamental similarities across species, but which is larger and more complex in humans. Proneural genes, encoding basic helix–loop–helix transcription factors (TFs), are evolutionarily conserved drivers of neurogenesis from fly to human ...
Lakshmy Vasan   +3 more
wiley   +1 more source

Cdk1 Controls Global Epigenetic Landscape in Embryonic Stem Cells.

open access: yesMolecules and Cells, 2020
W. Michowski   +28 more
semanticscholar   +1 more source

High‐throughput screening identifies a previously undescribed checkpoint controlling mitotic progression in response to DNA damage

open access: yesThe FEBS Journal, EarlyView.
DNA damage can cause mitotic delay to allow DNA repair to occur. However, the mechanism underlying this is unclear. Here, we show that in response to cells entering mitosis with DNA damage, SOD1 restrains PP2a activity via oxidation of cysteine residues at the active site. This leads to a reduction in PP2a activity at the mitotic kinetochore, resulting
Nan Li   +7 more
wiley   +1 more source

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