Results 11 to 20 of about 5,523 (171)

Prosthodontic rehabilitation of dentinogenesis imperfecta

open access: yesContemporary Clinical Dentistry, 2011
Dentinogenesis imperfecta and its prosthodontic management is a challenging task. Treatment protocol varies according to clinical case. Although various reports in the literature suggest general guidelines for treatment planning, the present case report ...
Anil Goud, Saee Deshpande
doaj   +2 more sources

Enzymes, Dentinogenesis and Dental Caries: a Literature Review

open access: yeseJournal of Oral Maxillofacial Research, 2014
Objectives: Search in PubMed with keywords “enzymes, dentinogenesis, and dental caries” revealed only 4 items, but when combined with “enzymes, osteogenesis, and osteoporosis” as high as 404 items resulted.
Markku Larmas, George K. B. Sándor
doaj   +2 more sources

Dentinogenesis imperfecta: A case report

open access: yesJournal of Indian Society of Pedodontics and Preventive Dentistry, 2008
Dentinogenesis imperfecta is an autosomal dominant disorder of tooth development characterized by the presence of opalescent dentin, resulting in a dusky blue to brownish discoloration of the teeth.
Subramaniam P, Mathew S, Sugnani S
doaj   +1 more source

Regenerative Endodontic Treatment in Dentinogenesis Imperfecta-Induced Apical Periodontitis

open access: yesCase Reports in Dentistry
Pulp involvement of immature permanent teeth with dentinogenesis imperfecta is challenging and could lead to extraction. A case of dentinogenesis imperfecta-induced periapical periodontitis of an immature permanent tooth was treated with regenerative ...
Ying Liao, Ting Pan, Xianghui Xing
doaj   +2 more sources

Promoting dentinogenesis of DPSCs through inhibiting microRNA-218 by using magnetic nanocarrier delivery

open access: yesJournal of the Formosan Medical Association, 2019
Purpose: The purposes of this study are to explore the roles of microRNA-218 (miR-218) delivered by a newly designed magnetic nanocarrier: GCC-Fe3O4 (GCC-Fe) in dentinogenesis potentials of human dental pulp stem cells (DPSCs).
Karen Chang   +3 more
doaj   +2 more sources

Chromatin Remodeller BRD9 Orchestrates Odontoblastic Differentiation via Coordinating RUNX2-KLF4. [PDF]

open access: yesCell Prolif
During odontoblast lineage commitment, the chromatin remodeller BRD9 acts as a critical epigenetic coordinator, orchestrating the chromatin landscape to facilitate synergistic binding of key transcription factors RUNX2 and KLF4 to target loci for odontogenesis.
Zeng W   +8 more
europepmc   +2 more sources

Multidisciplinary Oral Rehabilitation in Osteogenesis Imperfecta: 18-Year-Old Case Report. [PDF]

open access: yesClin Case Rep
ABSTRACT Osteogenesis imperfecta (OI) is a hereditary connective tissue disorder characterized by bone fragility and Type I collagen defects. Although dentinogenesis imperfecta (DI) is a classic manifestation, patients with OI may experience significant dental deterioration even in its absence due to inherent dentinal weakness.
Mozafari PM   +3 more
europepmc   +2 more sources

Dentinogenesis imperfecta: The Genetic Causes and Outcomes [PDF]

open access: yes, 2021
Dentinogenesis imperfecta (DI) is a genetic disorder characterized by dentin discoloration, tooth development irregularities, and decreased tooth strength. This autosomal dominant disorder is identified in individuals of all ages.
Johnson, Sydney P, Ms., Sydney Johnson
core   +1 more source

Cellular Dynamics and Dentine Bridge Formation Following Direct Pulp Capping With MTA and Biodentine in a Murine Model. [PDF]

open access: yesInt Endod J
ABSTRACT Aim Formation of a mineralized dentine bridge is the hallmark of successful direct pulp capping (DPC). Mineral trioxide aggregate (MTA) and Biodentine are widely used capping materials, but in vivo evidence comparing their regenerative potential remains inconclusive. This study used a controlled murine model to compare dentine bridge formation
Sajdeya A   +4 more
europepmc   +2 more sources

Analysis of Genetic Factors in a Family With Short Stature. [PDF]

open access: yesMol Genet Genomic Med
We identified pathogenic variants of two different genes, COL1A2 and GH1, in a family with short stature through trio‐WES, thereby providing a clear genetic diagnosis for this family. ABSTRACT Background To elucidate the genetic underpinnings of short stature in a familial cohort of five individuals.
Zhang S, Zuo T, Deng Y, Zhao D, Liao L.
europepmc   +2 more sources

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