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Physiology of GIP - A Lesson from GIP Receptor Knockout Mice
Hormone and Metabolic Research, 2004A much greater insulin response is observed after oral glucose load than after intravenous injection of glucose. The hormonal factor(s) implicated as transmitters of signals from the gut to pancreatic beta-cells was referred to incretin; gastric inhibitory polypeptide or glucose-dependent insulinotropic polypeptide (GIP) is identified as one of the ...
Y, Yamada, Y, Seino
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Physiology and Pathophysiology of GIP
1978Two major physiological roles for GIP have been established. An enterogastrone-like action in dog, in which it has been shown that physiological doses of the hormone will inhibit gastric acid secretion stimulated by histamine, gastrin or insulin hypoglycaemia (1) and an insulinotropic action of the hormone in both dog and man (2,3).
J C, Brown +4 more
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Clinical aspects of GIP secretion
Acta Diabetologica Latina, 1982The gastric inhibitory polypeptide (GIP) is the main hormone of the incretin type acting on the entero-insular axis. It is released after fat, glucose or meal ingestion. The variations of this secretion are described in obesity and in some pancreatic and gastrointestinal diseases: it is increased in maturity onset diabetes mellitus, obesity or duodenal
B, Beck, C, Villaume, G, Debry
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[Gastric inhibitory polypeptide (GIP) and GIP receptor (GIPR)].
Nihon rinsho. Japanese journal of clinical medicine, 1996Gastric inhibitory polypeptide, originally isolated from porcine intestine, is a gastrointestinal hormone belonging to the vasoactive intestinal peptide (VIP)/glucagon/secretin family. GIP consists of 42 amino acid residues which is derived by proteolytic processing of a GIP precursor.
K, Yasuda, Y, Seino
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GIP analogues augment bone strength by modulating bone composition in diet-induced obesity in mice
Peptides, 2020Sagar Vyavahare +2 more
exaly