Results 171 to 180 of about 34,963 (198)

GPI-Linked Proteins Do Not Transfer Spontaneously from Erythrocytes to Liposomes. New Aspects of Reorganization of the Cell Membrane

Biochemistry, 2000
Exposure of cells to liposomes results in the release of integral membrane proteins. However, it is still controversial whether the release is due to spontaneous protein transfer from cells to liposomes or shed vesicles released from cells. We investigated this issue in an erythrocyte-liposome system by examining the location of acetylcholinesterase ...
K, Suzuki, Y, Okumura
openaire   +2 more sources

Two distinct patterns of glycosylphosphatidylinositol (GPI) linked protein deficiency in the red cells of patients with paroxysmal nocturnal haemoglobinuria

British Journal of Haematology, 1992
Summary.We have studied three glycosylphosphatidylinositol (GPI) linked proteins on the erythrocytes of 14 patients with paroxysmal nocturnal haemoglobinuria (PNH). The pattern observed was bimodal in 12 of the patients and trimodal in two. Ten patients had a red cell population with normal CD59 antigen (membrane inhibitor of reactive lysis, MIRL ...
P, Hillmen, J M, Hows, L, Luzzatto
openaire   +2 more sources

Mutations in the PIG‐A gene causing partial deficiency of GPI‐linked surface proteins (PNH II) in patients with paroxysmal nocturnal haemoglobinuria

British Journal of Haematology, 1994
SummaryParoxysmal nocturnal haemoglobinuria (PNH) is due to the absence or marked reduction of glycan phosphatidylinositol (GPI)‐anchored proteins on the surface of blood cells. Affected patients may have a population of red blood cells that are completely deficient (PNH III) or partially deficient (PNH II) in these proteins, or they may have both. PNH
M, Bessler, P J, Mason, P, Hillmen, L, Luzzatto   +3 more
openaire   +2 more sources

Mice with Blood Cells Deficient in GPI-linked Proteins — The Generation of a Murine Model to Investigate the Pathogenesis and Pathophysiology of Paroxysmal Nocturnal Hemoglobinuria (PNH)

2003
Clinical characteristics of PNH include anemia due to intravascular hemolysis and hemoglobinuria, complications caused by bone marrow failure, and recurrent thrombosis. Despite the cloning of the Piga gene and the recent insights into the molecular lesion that causes the loss of glycosyl phosphatidylinositol-(GPI) linked proteins, much of the ...
Monica Bessler, Shashikant Kulkarni, Marek Jasinski, Anne Schaefer, Peter Keller, Gabi Tremml   +5 more
openaire   +1 more source

Expression of GPI-linked Green Fluorescent Protein on the surface of CHO cells

Biochemical Society Transactions, 2000
S. Hiscox, M. B. Hallett, C. W. van den Berg   +2 more
openaire   +1 more source

Post-ER degradation of misfolded GPI-anchored proteins is linked with microautophagy

Current Biology, 2021
Leticia Lemus, Maya Schuldiner, Lihi Gal   +2 more
exaly  

Profiling Glycosylphosphatidylinositol (GPI)-Interacting Proteins in the Cell Membrane Using a Bifunctional GPI Analogue as the Probe

Journal of Proteome Research, 2023
Zhongwu Guo, Sixue Chen, Chuwei Lin
exaly  

Growth factor-induced release of a glycosyl-phosphatidylinositol (GPI-) linked protein from HEp-2 carcinoma cells and human syncytiotrophoblast

Biochemical Society Transactions, 1991
J M, Roberts, P, Kenton, P M, Johnson
openaire   +2 more sources

Mechanisms of Sperm Acquisition of Glycosyl-phosphatidylinositol (GPI)-linked Epididymal Proteins.

Biology of Reproduction, 2008
openaire   +1 more source

Orientational Preferences of GPI-Anchored Ly6/uPAR Proteins

International Journal of Molecular Sciences, 2023
Ekaterina Lyukmanova, Anton Chugunov, Zakhar Shenkarev   +2 more
exaly