Transcriptional profiling of Hutchinson-Gilford progeria patients identifies primary target pathways of progerin. [PDF]
NucleusVidak S, Kim S, Misteli T.
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LAV-BPIFB4 reverses progeria-associated cardiac aging by restoring diastolic function and reducing senescence. [PDF]
Int J Biol SciChae U +4 more
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A study on the nutritional status and body composition of children with Hutchinson-Gilford progeria syndrome. [PDF]
Orphanet J Rare DisYu Q, Wang J, Fu H, Mao J.
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Lonafarnib Clinical Trials Demonstrate Uncoupling of the Muscle-Bone Unit in Hutchinson-Gilford Progeria Syndrome. [PDF]
J Bone Miner ResKreienkamp RJ +8 more
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Harnessing the HGP for Public Health [PDF]
Environmental Health Perspectives, 2004 openaire +1 more source
miR-140-5p Overexpression Contributes to Oxidative Stress and Mitochondrial Dysfunction in Hutchinson-Gilford Progeria Syndrome Fibroblasts Through NRF2 Pathway. [PDF]
Aging CellToury L +13 more
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Local metastatic expansion versus secondary intra-organ dissemination: two causes of neurological death explained by fundamentally different metastatic colonization patterns. [PDF]
Mol CancerKomljenovic D +14 more
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Examination of histopathological growth patterns of liver metastases in a retrospective, consecutive, single-center, cohort study. [PDF]
Pathol Oncol ResSejben A, Abbasi P, Pósfai B, Lantos T.
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A longevity-associated variant of the human BPIFB4 gene prevents diastolic dysfunction in progeria mice. [PDF]
Signal Transduct Target TherQiu Y +4 more
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