MetInfilt: A prospective trial highlighting the importance of the histological growth pattern in brain metastases. [PDF]
Proescholdt MA +15 more
europepmc +1 more source
Analysis of Beta-Dystroglycan in Different Cell Models of Senescence. [PDF]
Jimenez-Gutierrez GE +8 more
europepmc +1 more source
Involvement of NRF2 and AMPK signaling in aging and progeria: a digest. [PDF]
Petsouki E +4 more
europepmc +1 more source
Development and validation of a computed tomography-based radiomics-clinical model to preoperatively predict high-grade patterns within lung invasive adenocarcinoma. [PDF]
Zhang X +7 more
europepmc +1 more source
Enhancing the intraoperative identification of high-grade patterns in invasive lung adenocarcinoma via radiomics. [PDF]
Sun Y +9 more
europepmc +1 more source
A High Throughput Phenotypic Screening reveals compounds that counteract premature osteogenic differentiation of HGPS iPS-derived mesenchymal stem cells [PDF]
Hutchinson-Gilford progeria syndrome (HGPS) is a rare fatal genetic disorder that causes systemic accelerated aging in children. Thanks to the pluripotency and self-renewal properties of induced pluripotent stem cells (iPSC), HGPS iPSC-based modeling ...
Nicolas Levy, Lino Ferreira
exaly +5 more sources
Role of progerin-induced telomere dysfunction in HGPS premature cellular senescence [PDF]
Hutchinson-Gilford Progeria Syndrome (HGPS) is a premature-aging syndrome caused by a dominant mutation in the gene encoding lamin A, which leads to an aberrantly spliced and processed protein termed progerin.
Stuart Aaronson +2 more
exaly +3 more sources
Presence and distribution of progerin in HGPS cells is ameliorated by drugs that impact on the mevalonate and mTOR pathways [PDF]
Hutchinson–Gilford progeria syndrome (HGPS) is a rare, premature ageing syndrome in children. HGPS is normally caused by a mutation in the LMNA gene, encoding nuclear lamin A.
Evgeny Makarov +2 more
exaly +4 more sources

