Results 1 to 10 of about 504,948 (315)

Renal Denervation for Hypertension

Current Problems in Cardiology, 2014
Hypertension is the leading cause of mortality worldwide. Effective therapies are needed for resistant hypertension, a common condition characterized by inadequate blood pressure control despite 3 or more medications, which is associated with increased cardiovascular mortality.
Mackram F, Eleid, Gary L, Schwartz, Rajiv, Gulati   +2 more
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Mechanisms of renal hypertension and renal contribution to primary hypertension

Steroids, 1993
Investigators in hypertension have extensively evaluated the mechanisms of hypertension as first described by Goldblatt in his classic clipped kidney models. Although renovascular hypertension appears to affect only 2-4% of the population referred for diagnostic studies of hypertension, our understanding of renovascular hypertension has broadened from ...
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The Renal Medulla and Hypertension

Hypertension, 1995
Abstract We review evidence supporting the conclusion that renal dysfunction underlies the development of all forms of hypertension in humans and experimental animals. Indexes of global renal function are generally normal in the early stages of most genetic forms of hypertension, but renal function is clearly impaired in long ...
A W, Cowley, D L, Mattson, S, Lu, R J, Roman   +3 more
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Iatrogenic Renal Hypertension

Archives of Surgery, 1974
Delayed development of renal vascular hypertension secondary to surgical ligation of a major branch of the renal artery occurred. The blood pressure returned to normal levels after surgical removal of the ischemic section of the kidney. This case reiterates the importance of maintaining complete vascular supply to kidneys during renal surgery and ...
J L, McCormack, R C, Bain, G M, Kenny, R, Tocantins   +3 more
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Hypertension and Renal Disease

Veterinary Clinics of North America: Small Animal Practice, 1996
Systemic hypertension is commonly associated with chronic renal failure in dogs and cats. Consequences of systemic hypertension are manifested by pathologic changes involving the eyes, heart, central nervous system, and/or kidneys. These changes may be prevented or reversed by diagnosing and treating systemic hypertension.
J W, Bartges, A M, Willis, D J, Polzin
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Renal function in hypertension

The American Journal of Medicine, 1988
Hypertension certainly accelerates the age-related changes in renal structure and function, mainly in the glomerulus. Both sodium handling and the renin-angiotensin system are involved in the aging process. Among the available classes of antihypertensive agents, the eventual deterioration of renal function induced by angiotensin-converting enzyme ...
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Renal Medulla in Hypertension

Hypertension
Studies have found that blood flow to the renal medulla is an important determinant of pressure-natriuresis and the long-term regulation of arterial pressure. First, a brief review of methods developed enabling the study of the medullary circulation is presented. Second, studies performed in rats are presented showing medullary blood flow plays a vital
Allen W. Cowley, Richard J. Roman, David L. Mattson, Kleber G. Franchini, Paul M. O’Connor, Ayako Makino, Norman E. Taylor, Louise C. Evans, Takefumi Mori, Jeffrey G. Dickhout, Chunhua Jin, Noriyuki Miyata, Kazushige Nakanishi, Mátyás Szentiványi, Frank Park, Meredith M. Skelton, Theresa Kurth, Satoshi Shimada   +17 more
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Renal afferents and hypertension

Current Hypertension Reports, 2002
The kidney and the autonomic nervous system are linked through renal nerves. Activation of efferent renal sympathetic nerves leads to changes in renal vascular resistance, renin release, and Na(+) and water retention. Evidence also exists indicating that the kidney is not just a target organ of sympathetic activity, but also acts as a sensor.
John, Ciriello, Cleusa V R, de Oliveira
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MECHANISM OF RENAL HYPERTENSION

The Lancet, 1976
Renal hypertension of the two-kidney type is divided into three stages. In the first, hypertension results from the vasoconstrictor effect of angiotensin II. This persists to some extent in the second phase but there is in addition a slow-developing pressor effect, also resulting from angiotensin II and probably attributable to sodium. In the first two
J.J. Brown, D.L. Davies, J.J. Morton, J.I.S. Robertson, V. Cuesta, A.F. Lever, P.L. Padfield, P. Trust, G. Bianchi, M.A.D. Schalekamp   +9 more
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Renal Hypertension

JAMA, 1963
A N, BREST, J H, MOYER
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