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Pre- and Perinatal Ischemia-Hypoxia, the Ischemia-Hypoxia Response Pathway, and ADHD Risk [PDF]
Behavior Genetics, 2016 This review focuses on how measured pre- and perinatal environmental and (epi)genetic risk factors are interrelated and potentially influence one, of many, common developmental pathway towards ADHD. Consistent with the Developmental Origins of Health and
Jessica A Kaczorowski +2 more
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Regional Changes in Metabolism in Hypoxia-Ischemia
1977The purpose of this study is to describe regional metabolic changes following an episode of cerebral ischemia in the cat. Regional characterization of metabolic derangements is imperative because it is unlikely that cerebral blood flow is uniformly reduced in most hypoxic-ischemic episodes.1, 2, 3 Furthermore, neurons in discrete anatomic regions such ...
F A, Welsh +3 more
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Transgenerational effects of neonatal hypoxia‐ischemia in progeny
International Journal of Developmental Neuroscience, 2013AbstractNeonatal hypoxia‐ischemia (HI) affects 60% of low birth weight infants and up to 40% of preterm births. Cell death and brain injury after HI have been shown to cause long‐lasting behavioral deficits. By using a battery of behavioral tests on second generation 3‐week‐old rodents, we found that neonatal HI is associated with behavioral outcomes ...
Smitha K, Infante +2 more
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New concepts in perinatal hypoxia ischemia encephalopathy
Current Neurology and Neuroscience Reports, 2008This article summarizes recent insights into perinatal hypoxic-ischemic brain injury in the neonate. Before effective treatments can be offered, diagnosis, timing, and an understanding of the pathogenesis are imperative. The analysis of appropriate animal models is also summarized in this review.
Joseph, Scafidi, Vittorio, Gallo
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Oxidant Mechanisms in Neonatal Hypoxia-Ischemia
Developmental Neuroscience, 2001The neonatal brain appears to be selectively vulnerable to oxidative stress. Several potential mechanisms associated with altered reactive oxygen species metabolism would explain the increased susceptibility. They include increased accumulation of hydrogen peroxide with subsequent neurotoxicity. This enhanced neurotoxicity from H<sub>2</sub>
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Impairment of perinatal hypoxia–ischemia to the preterm brainstem
Journal of the Neurological Sciences, 2009Hypoxia-ischemia is a major perinatal problem that results in severe damage to the newborn brain. This study assessed functional integrity of the brainstem at term in preterm infants after perinatal hypoxia-ischemia to shed light on the influence of hypoxia-ischemia on the preterm brainstem.
Ze D, Jiang +3 more
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Autophagy in hypoxia-ischemia induced brain injury
The Journal of Maternal-Fetal & Neonatal Medicine, 2012Autophagy is an endogenous tightly regulated process responsible for the degradation of damaged and dysfunctional cellular organelles and protein aggregates. Emerging data indicate a strong and complex interaction among autophagy, apoptosis and necrosis. We studied these interactions in a neonatal model of hypoxia-ischemia (HI).
BALDUINI, WALTER +2 more
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Hypoxia/Ischemia and the pH Paradox
1996Hydrogen ions play an important role in cellular processes. There are intimate links between energy metabolism and the control of cell and tissue acid/base balance. Control of this balance is threatened or lost during severe hypoxia or ischemia. Re-establishment of pH balance must occur before the tissue can be considered to have returned to normal ...
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Progressive Multicystic Encephalopathy: Is There More Than Hypoxia-Ischemia?
Journal of Child Neurology, 2007Progressive multicystic encephalopathy following prenatal or perinatal hypoxia-ischemia is a well-described phenomenon in the literature. The authors report on a term infant with a devastating encephalopathy and severe neuronal dysfunction immediately after delivery without a known antecedent of prenatal or perinatal hypoxia or distress.
Garten, Lars +7 more
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The role of hypoxia/ischemia preconditioning in ischemic stroke
NeuroscienceIschemic stroke can cause damage to neurons, resulting in neurological dysfunction. The main treatments in the acute phase include intravenous thrombolysis, endovascular stent-assisted vascular thrombectomy and antiplatelet therapy. Due to the limitations of the time window and the risk of early intracranial hemorrhage, finding active treatment plans ...
Longni, Zhu +9 more
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