Results 141 to 150 of about 10,434 (197)

Function and mechanism of cancer-testis antigen CT63 in chronic myeloid leukemia

open access: yesShanghai Jiaotong Daxue xuebao. Yixue ban
Objective·To explore the effects of the cancer-testis antigen (CTA) family member CT63 on proliferation, differentiation, and tumorigenicity of chronic myeloid leukemia (CML) cells, and uncover the underlying molecular mechanisms.Methods·The link between
KONG Ruxin   +3 more
doaj   +1 more source

Synergistic effect of the combination of nanoparticulate Fe3O4 and Au with daunomycin on K562/A02 cells

open access: yesInternational Journal of Nanomedicine, 2008
Bao-An Chen1, Yong-Yuan Dai1, Xue-Mei Wang2, Ren-Yun Zhang2, Wen-Lin Xu3, Hui-Ling Shen3, Feng Gao1, Qian Sun1, Xiao-Jing Deng1, Jia-hua Ding1, Chong Gao1, Yun-Yu Sun1, Jian Cheng1, Jun Wang1, Gang Zhao1, Ning-Na Chen11Department of Hematology, The ...
Bao-An Chen   +5 more
doaj  

Co-culture of human bone marrow derive dmesenchymal stem cells with k562 cells conferred resistance to k562 cells against cytarabine treatment [PDF]

open access: yes
Background: Human mesenchymal stem cells (H-MSCs) are population of non-hematopoietic cells that have many applications in medicine most notably in cell therapy.
ملک زاده, ودود   +3 more
core  

H-ferritin uptake by K562 cells.

open access: yes, 2015
(A) Confocal micrographs of AF488-labeled H-ferritin (HFt) and holo-transferrin (Tf). Cells were incubated with 50 μg/ml (110 nM) AF488-labeled human recombinant HFt or 50 μg/ml (625 nM) holo-Tf for 1 h at 37°C. Images are representative results of three
Tatsuki Uchiyama (803736)   +8 more
core   +1 more source

ACM induced erythroid differentiation of K562 cells through p38MAPK pathway.

open access: yes, 2013
(A) K562 cells were treated with or without (control) 10 nM ACM, 2 µM SB202190 (SB), or ACM plus SB202190 for 3 days. Cell lysates were immunoprecipitated with anti-phospho-p38MAPK antibody.
Chih-Wei Chen (403470)   +4 more
core   +1 more source

Apoptosis and mitotic slippage following drug intervention in leukaemia cells

open access: yes, 2011
PhDThe response of leukaemia cells to therapeutic agents includes cell cycle arrest and apoptosis. The former response is useful in retarding disease progression, but induction of the latter is essential for disease eradication.
Omar, Najood Amer
core  

Expressions of P-glycoprotein, survivin and CD147 molecules on multidrug resistance leukemic cell line K562/Adr

open access: yesJournal of Associated Medical Sciences, 2015
Introduction: Leukemia is a hematologic malignant disease. To date, chemotherapy is the most effective method for leukemia treatment. However, multidrug resistance (MDR) is a major obstacle of leukemia treatment failure.
Aoranit Somno   +2 more
doaj  

Plenary Abstracts Session & Oral Presentations

open access: yes
HemaSphere, Volume 10, Issue S1, June 2026.
wiley   +1 more source

Poster Sessions

open access: yes
HemaSphere, Volume 10, Issue S1, June 2026.
wiley   +1 more source

Home - About - Disclaimer - Privacy