Results 201 to 210 of about 17,474 (245)

β-arrestin1 protects intestinal tight junction through promoting mitofusin 2 transcription to drive parkin-dependent mitophagy in colitis. [PDF]

Gastroenterol Rep (Oxf)
Wu S, Liu H, Yi J, Xu M, Jiang J, Tao J, Wu B.   +6 more
europepmc   +1 more source

Erratum: Erratum: Novel putative interactors of FZO-1/mitofusin 2 identified using large-scale yeast two-hybrid screening in C. elegans. [PDF]

MicroPubl Biol, 2022
europepmc   +1 more source

Corrigendum to "Inhibition of Mitofusin-2 Promotes Cardiac Fibroblast Activation via the PERK/ATF4 Pathway and Reactive Oxygen Species". [PDF]

Oxid Med Cell Longev
europepmc   +1 more source

Mitofusin 2 Variant Presenting With a Phenotype of Multiple System Atrophy of Cerebellar Subtype. [PDF]

Neurol Genet
Elbert A, Dixon K, Shen Y, Hamilton S, Boerkoel CF, Jones SJ, Kanungo AK.   +6 more
europepmc   +1 more source

Endosomal RFFL ubiquitin ligase regulates mitochondrial morphology by targeting mitofusin 2.

J Cell Sci
Narendradev ND, Ravindran R, Jain P, Chaudhary S, Velikkakath AKG, Sudharman A, Janardhanan A, Nag TC, Yadav SC, Srinivasula SM.   +9 more
europepmc   +1 more source

Acetate ameliorates ovarian mitochondrial dysfunction in letrozole-induced polycystic ovarian syndrome rat model by improving mitofusin-2. [PDF]

J Physiol Sci
Olaniyi KS, Areloegbe SE.
europepmc   +1 more source

Prenatal hormone stress triggers embryonic cardiac hypertrophy outcome by ubiquitin-dependent degradation of mitochondrial mitofusin 2. [PDF]

iScience
Yan CY, Ye Y, Mu HL, Wu T, Huang WS, Wu YP, Sun WY, Liang L, Duan WJ, Ouyang SH, Huang RT, Wang R, Sun XX, Kurihara H, Li YF, He RR.   +15 more
europepmc   +1 more source

Regulation of mitochondria-dynactin interaction and mitochondrial retrograde transport in axons [PDF]

, 2017
Drerup, Catherine M, Herbert, Amy L, Monk, Kelly R, Nechiporuk, Alex V   +3 more
core   +1 more source

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Evaluation of the role of mitofusin‐1 and mitofusin‐2 in periodontal disease

Journal of Periodontology, 2023
AbstractBackgroundMitochondria and endoplasmic reticulum are key cellular organelles and create contact sites (mitochondria‐endoplasmic reticulum contact [MERC]), which plays a major role in calcium metabolism, apoptotic processes, and inflammation.
Ömer Alperen Kırmızıgül, Arife Sabanci, Faruk Dişli, Sedat Yıldız, Michael R. Milward, Kübra Aral   +5 more
openaire   +2 more sources