Results 111 to 120 of about 16,176 (222)

Protective effect of N-acetylcysteine on MK-801-induced testicular oxidative stress in mice

open access: yes, 2019
Experimental studies indicate that MK-801 causes organ injury in schizophrenic mice testes although it is molecular mechanism has not been clearly defined. In this study, we investigated the probable protective effect of N-Acetylcysteine (NAC) against MK-
Ruhi Turkmen   +5 more
core   +1 more source

MK-801 does not protect against hypoxic-ischemic brain injury in piglets.

open access: yes, 1991
The excitatory amino acid inhibitor MK-801 has been shown in many animals species to protect against hypoxic-ischemic brain injury. We sought to determine whether hypoxic-ischemic injury to the newborn pig's brain could be prevented by the use of MK-801.
V Vig   +5 more
core   +1 more source

MK-801 interferes with the acquisition of amphetamine- and lithium-induced place conditioning

open access: yes, 1997
The place conditioning paradigm was used to assess the ability of the noncompetitive NMDA receptor antagonist, M- 801 to interfere with drug-place associations.
Toth, Jonathan Francis
core   +1 more source

Behavioral effects of MK-801 on reserpine-treated mice

open access: yes, 2002
The effects of dizocilpine (MK-801), a noncompetitive N-methyl-D-aspartate (NMDA) receptor antagonist, were studied on dopamine-related behaviors induced by reserpine treatments.
Andreazza, A. P.   +4 more
core   +1 more source

Effects of MK-801 on Cocaine Place Conditioning in Adolescent Female Rats

open access: yes, 2011
One of the most studied neurotransmitter systems in addiction is the mesolimbic dopamine pathway, which primarily uses dopamine to build a reward and learning mechanism. Glutamate and NMDA receptors are also used in the pathway. MK-801 is a NMDA receptor
Dhaliwal, Aman
core   +1 more source

Sustained MK-801 induced deficit in a novel probabilistic reversal learning task. [PDF]

open access: yesFront Pharmacol, 2022
Latuske P   +4 more
europepmc   +1 more source

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