Correction: Non-canonical cell death in neurodegeneration: emerging mechanisms and therapeutic Frontiers. [PDF]
Ercin N, Besli N, Beker M, Celik U.
europepmc +1 more source
MAFF Suppresses Necroptosis in Pancreatic Cancer via the GATA4-MLKL Axis. [PDF]
Zhou X, Zhao Z, Sun N, Zhang D.
europepmc +1 more source
OTUD7B exacerbates atherosclerosis by promoting RIPK1-dependent vascular smooth muscle cell necroptosis. [PDF]
Yu L, Wang Y, Li Y, Zhang Y.
europepmc +1 more source
Mitochondrial Dysfunction at the Crossroads of Necroptosis: Mechanisms, Molecular Mediators, and Therapeutic Opportunities. [PDF]
Wang F +9 more
europepmc +1 more source
Separation, Structural Elucidation, and Anticancer Activity of a Diastereoisomer of Betulonic Acid-Zidovudine Conjugates. [PDF]
Zhai J +11 more
europepmc +1 more source
Astragaloside IV mitigated cadmium-induced glandular gastric injury in ducks by inhibiting PANoptosis. [PDF]
Gao X +9 more
europepmc +1 more source
Mlkl knockout mice demonstrate the indispensable role of Mlkl in necroptosis [PDF]
National Basic Research Program of China (973 Program) [2009CB522201]; National Natural Science Foundation of China [91029304, 30830092, 30921005, 81061160512]; Hi-Tech Research and Development Program of China (863 program) [2012AA02A201]; 111 Project ...
Jiahuai Han, Zhe Huang, Zhirong Zhang
exaly +5 more sources
The Many Faces of MLKL, the Executor of Necroptosis [PDF]
Necroptosis is a recently discovered form of regulated cell death characterized by the disruption of plasma membrane integrity and the release of intracellular content. Mixed lineage kinase domain-like (MLKL) protein is the main player of this cell death
Abdelwahab, Y. +5 more
exaly +5 more sources
MLKL forms cation channels [PDF]
The mixed lineage kinase domain-like (MLKL) protein is a key factor in tumor necrosis factor-induced necroptosis. Recent studies on necroptosis execution revealed a commitment role of MLKL in membrane disruption. However, our knowledge of how MLKL functions on membrane remains very limited.
Huayi Wang, Zhaobing Gao, Xueqin Chen
exaly +8 more sources

