Results 201 to 210 of about 31,768 (245)

Volume changes in isolated myofibrils

Biochimica et Biophysica Acta (BBA) - Specialized Section on Biophysical Subjects, 1964
Volume changes in glycerol-extracted myofibrils were studied. The effect of pH, ion content and ATP concentration on the change in volume was determined. A volume change greater than that attributed to ATP dephosphorylation was observed. The magnitude of the volume change of the entire system was 10−3 cm3 whereas that due to dephosphorylation was 10−6 ...
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Binding of filamin isoforms to myofibrils

Journal of Muscle Research & Cell Motility, 2000
Two filamin isoforms were purified from bovine tissues and characterized. Muscle filamin and nonmuscle filamin had different SDS gel mobilities, proteolytic digestion patterns, myofibrillar binding distributions and myofibril binding affinities. The muscle specific filamin had an apparent molecular weight of 265 kDa and bound primarily to the Z-lines ...
W, Chiang, M L, Greaser
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Preparation of myosin rods from myofibrils

Biochemical and Biophysical Research Communications, 1973
Abstract A simple and fast procedure is described for producing myosin rods with a good yield. The method is based on the digestion of myofibrils by soluble papain. After purification by alcoholic precipitation and gel filtration, rod segments exhibited physical-chemical parameters identical to previously reported data.
C, Oriol, M F, Landon, N, van Thoai
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[2] Preparation of myofibrils

1982
Publisher Summary A variety of procedures are available for the preparation of myofibrils, and this chapter presents four of these that include myofibrils from fresh rabbit psoas muscle, myofibrils from glycerinated psoas muscle, myofibrils in bulk from fresh muscle, and myofibrils in bulk from stored muscle..
Peter J. Knight, John A. Trinick
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To the heart of myofibril assembly

Trends in Cell Biology, 2000
One of the most fascinating examples of cytoskeletal assembly is the myofibril, the contractile structure of striated (i.e. skeletal and cardiac) muscle. Myofibrils are composed of repeating contractile units known as sarcomeres, perhaps the most highly ordered macromolecular structures in eukaryotic cells.
C C, Gregorio, P B, Antin
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Adaptation in der Myofibrille

physiopraxis, 2004
Muskelkater – wodurch entsteht er? Muskelschaden oder Remodellierungsprozess der Myofibrillen? Und wie kann man ihn behandeln? Wissenschaftler aus aller Welt diskutieren Pathogenese und Therapie. Wilhelm E. Kraft hat für physiopraxis die aktuelle Literatur recherchiert und die Studienergebnisse auf den Punkt gebracht.
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Contraction-Band Formation in Barnacle Myofibrils

Science, 1964
Contractions induced by adenosine triphosphate, in myofibrils isolated from the barnacle, Balanus aquilia , were observed with a phase microscope. The formation of contraction bands was well under way before the A-band came into contact with the Z-membrane.
R J, BASKIN, G M, WIESE
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Passive force enhancement in single myofibrils

Pflügers Archiv - European Journal of Physiology, 2007
The purpose of this study was to gain further insight into passive force enhancement by testing whether passive force enhancement occurs in single myofibrils. Myofibrils (n = 6) isolated from rabbit psoas muscle were fixed at a sarcomere length of 2.4 microm, and then stretched passively and actively to a sarcomere length of 3.4 microm.
V, Joumaa, D E, Rassier, T R, Leonard, W, Herzog   +3 more
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Residual force depression in cardiac myofibrils

Journal of Biomechanics
History-dependent properties, such as residual force depression (rFD; i.e., the decrease in steady-state isometric force of a muscle, following active shortening, compared to the corresponding force of a purely isometric contraction) have been consistently observed in skeletal muscle.
Seong-Won, Han, Torri, Heiser, Venus, Joumaa, Walter, Herzog   +3 more
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Cardiomyopathies and Myofibril Abnormalities

2002
Cardiomyopathies are primary disorders of the myocardium in which systolic and/or diastolic dysfunction occurs (Towbin, 1993, 1999). These cardiac muscle diseases are classified based on phenotypic features (Towbin, 1993, 1999; Richardson et al., 1996) and include: (1) dilated cardiomyopathy (DCM), (2) hypertrophic cardiomyopathy (HCM), (3) restrictive
Jeffrey A. Towbin, Neil E. Bowles
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