Results 11 to 20 of about 2,754 (217)

The absence of early malignant changes in women subjected to Aquafilling breast augmentation on the basis of E-cadherin and N-cadherin immunohistochemical expression

open access: yesCentral European Journal of Immunology, 2023
The use of Aquafilling can be associated with a variety of health complications. The filler is an inflammatory process trigger at the site of tissue contact.
Michał Chalcarz, Jakub Żurawski
doaj   +1 more source

Vimentin is a potential prognostic factor for tongue squamous cell carcinoma among five epithelial-mesenchymal transition-related proteins. [PDF]

open access: yesPLoS ONE, 2017
We aimed to investigate the association of the expression levels of five epithelial-mesenchymal transition (EMT)-related proteins (Snail, Twist, E-cadherin, N-cadherin, and Vimentin) with tumorigenesis, pathologic parameters and prognosis in tongue ...
Pei-Feng Liu   +14 more
doaj   +1 more source

N‐cadherin promoter polymorphisms and risk of osteoarthritis [PDF]

open access: yesThe FASEB Journal, 2013
Osteoarthritis (OA) is the most common form of arthritis. It is characterized by cartilage destruction and bone remodeling, mediated in part by synovial fibroblasts (SFs). Given the functional significance of cadherins in these cells, we aimed at determining the role of genetic variants of N‐cadherin (CDH2)
Ruedel, Anke   +8 more
openaire   +3 more sources

Pathologic Proteolytic Processing of N-Cadherin as a Marker of Human Fibrotic Disease

open access: yesCells, 2022
Prior research has implicated the involvement of cell adhesion molecule N-cadherin in tissue fibrosis and remodeling. We hypothesize that anomalies in N-cadherin protein processing are involved in pathological fibrosis.
Paul Durham Ferrell   +3 more
doaj   +1 more source

E-cadherin can replace N-cadherin during secretory-stage enamel development.

open access: yesPLoS ONE, 2014
BackgroundN-cadherin is a cell-cell adhesion molecule and deletion of N-cadherin in mice is embryonic lethal. During the secretory stage of enamel development, E-cadherin is down-regulated and N-cadherin is specifically up-regulated in ameloblasts when ...
Xiaomu Guan   +3 more
doaj   +1 more source

Effects of N-cadherin silencing on the proliferation and migration of human dental pulp stem cells

open access: yes口腔疾病防治, 2022
Objective To investigate the effects of N-cadherin silencing on the proliferation and migration of human dental pulp stem cells (DPSCs) and to provide experimental evidence for DPSCs-based dental pulp regeneration.
DENG Zilong   +3 more
doaj   +1 more source

N-cadherin, spine dynamics, and synaptic function [PDF]

open access: yesfrontiers in Neuroscience, 2008
Dendritic spines are one-half (the postsynaptic half) of most excitatory synapses. Ever since the direct observation over a decade ago that spines can continually change size and shape, spine dynamics has been of great research interest, especially as a mechanism for structural synaptic plasticity.
Mysore, S., Tai, C., Schuman, E.
openaire   +4 more sources

Data on the effects of N-cadherin perturbation on the expression of type II cadherin proteins and major signaling pathways

open access: yesData in Brief, 2018
This article contains data related to the research article entitled, “A catenin-dependent balance between N-cadherin and E-cadherin controls neuroectodermal cell fate choices” (Rogers et. al., 2018) [1].
Crystal D. Rogers
doaj   +1 more source

N-cadherin mediates neuronal cell survival through Bim down-regulation. [PDF]

open access: yesPLoS ONE, 2012
N-cadherin is a major adhesion molecule involved in the development and plasticity of the nervous system. N-cadherin-mediated cell adhesion regulates neuroepithelial cell polarity, neuronal precursor migration, growth cone migration and synaptic ...
Elise C Lelièvre   +5 more
doaj   +1 more source

N-cadherin: stabilizing synapses [PDF]

open access: yesJournal of Cell Biology, 2010
Spines are sites of excitatory synapse formation in central neurons. Alterations in spine structure and function are widely believed to actively contribute to the cellular mechanisms of learning and memory. In this issue, Mendez et al. (2010. J. Cell Biol.
openaire   +2 more sources

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