Identification and Functional Characterization of Novel and Recurrent NTRK1 Variants in Chinese Families With Congenital Insensitivity to Pain With Anhidrosis: A Combined Clinical, Genetic, and Functional Study. [PDF]
Ren Y +9 more
europepmc +1 more source
The neural stem cell‑NSCLC axis: Molecular drivers, microenvironment crosstalk and emerging therapies (Review). [PDF]
Xu Y, Duan H, Lu P, Liu F.
europepmc +1 more source
Activation of Nerve Growth Factor signaling limits the response to lenvatinib in hepatocellular carcinoma. [PDF]
Xu M +16 more
europepmc +1 more source
A critical appraisal of the safety of bedinvetmab (Beransa), a canine antinerve growth factor monoclonal antibody. [PDF]
Yang X, Macarthur P.
europepmc +1 more source
Umbilical Cord Blood Gasometry and pH as Key Regulators of Growth Factor Expression Profile in Umbilical Cord-Derived Mesenchymal Stromal Cells (UC-MSCs). [PDF]
Przywara D +9 more
europepmc +1 more source
Tear biomarker changes and ocular surface recovery with low-level light therapy after cataract surgery: a double-masked randomized controlled clinical trial. [PDF]
Timofte-Zorila MM +13 more
europepmc +1 more source
Related searches:
NGF ligand alters NGF signaling via p75NTR and TrkA
Journal of Neuroscience Research, 2000Nerve growth factor (NGF) binds to two neurotrophin receptors: p75(NTR) and p140(Trk) (TrkA). Both receptors dimerize in response to NGF binding. TrkA homodimers and heteromers of TrkA and p75(NTR) promote cell survival whereas homodimers of p75(NTR) mediate apoptosis upon binding of NGF. The interaction between receptor and NGF can be inhibited either
O, Niederhauser +5 more
exaly +3 more sources
NGF/TrkA Signaling as a Therapeutic Target for Pain
Pain Practice, 2016Munetaka Hirose
exaly +2 more sources
The Emerging Therapeutic Role of NGF in Alzheimer’s Disease
Neurochemical Research, 2016Wei-Lin Jin, Jin Wei-Lin
exaly +2 more sources
ATP–NGF-complex, but not NGF, is the neuroprotective ligand
Neurochemistry International, 2011We have shown previously that nerve growth factor (NGF) requires only low nanomolar ATP concentrations in the cell culture medium to protect cortical rat neurons (CRN) from cellular damage induced by staurosporine (STS). We have also demonstrated before that NGF and other growth factors form stable non-covalent complexes with ATP.
Katja Bettina, Ferenz +3 more
openaire +2 more sources

