Results 241 to 250 of about 188,463 (312)

On-Demand Deconstructable Thermosets Through Cleavable Comonomer and Thermolatent Base Synergy. [PDF]

Angew Chem Int Ed Engl
Kouider S, Buchon L, Choulot L, Greuet P, Bratasanu J, Désirée P, Mauriello J, Maarek E, Clément JL, Charles L, Gastaldi E, Montarnal D, Soppera O, Gigmes D, Lefay C, Guillaneuf Y.   +15 more
europepmc   +1 more source

Riluzole shifts glial responses to protect synapses and memory in Aβ oligomer-treated rats. [PDF]

J Neuroinflammation
Min-Kaung-Wint-Mon, Kida H, Kimura R, Mitsushima D.   +3 more
europepmc   +1 more source

Shrinking of Extracellular Space During Metabolic Stress Accelerates Amyloid-β Aggregation. [PDF]

Biomolecules
De Oliveira LF, Karunarathne K, Zona D, Muschol M, Ullah G.   +4 more
europepmc   +1 more source

Conformality of atomic/molecular layer deposited lithium terephthalate thin films.

Nanoscale
Philip A, Madadi M, Jestilä JS, Karttunen AJ, Kinnunen J, Karppinen M.   +5 more
europepmc   +1 more source

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Unnatural oligomers and unnatural oligomer libraries

Current Opinion in Chemical Biology, 1997
Within the past few years, a variety of compounds that mimic biopolymers have been developed. All of these unnatural oligomers are prepared by iterative syntheses, which are amenable to combinatorial strategies. Peptides continue to be popular targets for mimicry, and there is growing interest in targeting oligosaccharides.
M J, Soth, J S, Nowick
openaire   +2 more sources

Amyloid oligomers: formation and toxicity of Aβ oligomers

The FEBS Journal, 2010
Alzheimer’s disease (AD) is an age‐related, progressive degenerative disorder that is characterized by synapse and neuron loss in the brain and the accumulation of protein‐containing deposits (referred to as ‘senile plaques’) and neurofibrillary tangles. Insoluble amyloid β‐peptide (Aβ) fibrillar aggregates found in extracellular plaques have long been
Masafumi, Sakono, Tamotsu, Zako
openaire   +2 more sources

Tau Oligomers

2019
The accumulation of tau filaments in neurons is a pathological hallmark of various neurodegenerative diseases, including Alzheimer's disease. However, it is not the filamentous aggregates themselves, but non-filamentous tau species, tau oligomer, that is thought to be the culprit in tau-mediated neurodegeneration.
Sumihiro, Maeda, Akihiko, Takashima
openaire   +2 more sources