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Is there a link between non-steroidal anti-inflammatory drugs and cardiovascular risk in patients with inflammatory arthritis? [PDF]
Atheroscler PlusZingler G, Baerwald C.
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Endothelial triple-pathway vasorelaxation as an adjunctive strategy in resistant hypertension. [PDF]
J HypertensMadeddu P +3 more
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Angiology, 1989
Hemodialysis performed with prostacyclin (5 ng/kg/min) as a sub stitute for heparin was studied in 10 patients. The subjects were studied during heparin perfusion alone and during heparin perfusion together with prostacyclin. The authors inves tigated the effect of two heparin regi mens (regimen I: 2,000 U/hr and reg imen II: 500 U/hr) upon plasma anti
CAMICI, MARCELLO, EVANGELISTI L.
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Hemodialysis performed with prostacyclin (5 ng/kg/min) as a sub stitute for heparin was studied in 10 patients. The subjects were studied during heparin perfusion alone and during heparin perfusion together with prostacyclin. The authors inves tigated the effect of two heparin regi mens (regimen I: 2,000 U/hr and reg imen II: 500 U/hr) upon plasma anti
CAMICI, MARCELLO, EVANGELISTI L.
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European Journal of Obstetrics & Gynecology and Reproductive Biology, 1984
Much attention has been focused in recent years on two particular metabolites of arachidonic acid, prostacyclin (PGI,) and thromboxane A, (TXA,). The prostanoid PGI, is the main product of arachidonic acid in all arteries and veins so far tested. It is a potent vasodilator and the strongest endogeneous inhibitor of platelet aggregation yet discovered ...
B, Spitz +3 more
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Much attention has been focused in recent years on two particular metabolites of arachidonic acid, prostacyclin (PGI,) and thromboxane A, (TXA,). The prostanoid PGI, is the main product of arachidonic acid in all arteries and veins so far tested. It is a potent vasodilator and the strongest endogeneous inhibitor of platelet aggregation yet discovered ...
B, Spitz +3 more
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1991
During extracorporeal circulation blood interaction with foreign surfaces leads to activation of cells i.e. throm bocytes and white blood cells (1).
CAMICI, MARCELLO, EVANGELISTI L.
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During extracorporeal circulation blood interaction with foreign surfaces leads to activation of cells i.e. throm bocytes and white blood cells (1).
CAMICI, MARCELLO, EVANGELISTI L.
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Respiratory Care, 2004
Two prostacyclins (prostaglandin E(1) and prostaglandin I(2)) are potent vasodilators. Aerosolized prostacyclins reduce pulmonary artery pressure, improve right heart function, and increase arterial oxygenation by improving ventilation/perfusion matching. This report describes aerosolized prostacyclins and compares them to inhaled nitric oxide.
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Two prostacyclins (prostaglandin E(1) and prostaglandin I(2)) are potent vasodilators. Aerosolized prostacyclins reduce pulmonary artery pressure, improve right heart function, and increase arterial oxygenation by improving ventilation/perfusion matching. This report describes aerosolized prostacyclins and compares them to inhaled nitric oxide.
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2013
Prostacyclins have a favourable pharmacological profile for treatment of pulmonary hypertension as they possess vasodilative, antiproliferative, antiaggregatory, and anti-inflammatory properties that may compensate the main pathologic changes in the small pulmonary arteries.
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Prostacyclins have a favourable pharmacological profile for treatment of pulmonary hypertension as they possess vasodilative, antiproliferative, antiaggregatory, and anti-inflammatory properties that may compensate the main pathologic changes in the small pulmonary arteries.
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Lipoproteins and Prostacyclin Stability
Seminars in Thrombosis and Hemostasis, 1993PGI2 is an important local mediator keeping circulating blood cells apart from the vessel wall, thus regulating hemostasis. Alterations of locally available amounts of this compound may be associated with either bleeding or thrombotic events. Factors influencing synthesis, transmission, and degradation coregulate the amount of biologically active PGI2 ...
C, Pirich +3 more
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Prostacyclin and vascular disease
Philosophical Transactions of the Royal Society of London. B, Biological Sciences, 1981Abstract We hypothesize that prostacyclin (PGI2) is an anti-atherosclerotic hormone and that atherosclerosis develops when endothelial PGI2 synthetase is inhibited by lipid peroxides. Serum lipid peroxides occur in low-density lipoproteins (LDL).
R J, Gryglewski +3 more
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