Results 131 to 140 of about 14,486 (179)
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Restriction of receptor mobility and the agglutination of cells by concanavalin a
Experimental Cell Research, 1975Summary Transformed hamster fibroblasts with a high ATP content which are not agglutinated by concanavalin A (ConA) can be induced to agglutinate by decreasing their ATP content, mixing of glutaraldehyde prefixed ConA-coated cells with unfixed uncoated cells, or mixing of unfixed ConA-saturated cells with uncoated cells.
I, Vlodavsky, L, Sachs
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The effects of concanavalin a on the mobility of lymphocyte surface receptors
Experimental Cell Research, 1973Abstract It has been found that concanavalin A (Con A) bound to the lymphocyte surface can either induce cap formation or inhibit cap formation of various receptors including those for Con A itself. The expression of these antagonistic activities is highly dependent on the conditions under which cells are incubated with Con A.
I, Yahara, G M, Edelman
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Interaction of concanavalin A with lactogenic receptors in isolated membranes
Archives of Biochemistry and Biophysics, 1982Abstract Specific binding of lactogenic hormones to their receptors in membranes from lactating mouse liver is inhibited by concanavalin A (Con A). Binding to solubilized receptors is not affected by the presence of Con A in the binding reaction. However, these solubilized binding proteins are retained on Con A-Sepharose columns.
Bhattacharya, A, Vonderhaar, B K
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Receptors for concanavalin A cluster at the front of polarized neutrophils
Nature, 1980Polymorphonuclear neutrophils (PMN) can recognize, engulf and kill microorganisms and are thus an important host defence against infection. The initial encounter between a microbe and the neutrophil takes place at the neutrophil surface. Receptors, including those for the Fc end of the immunoglobulin G (ref.
D L, Weinbaum +2 more
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Concanavalin a Binding Receptors on Trypanosoma cruzi Amastigotes
The Journal of Parasitology, 1980501-505) as adapted by Garcia et al. (1978, Arch. Biochem. Biophys. 188: 315-322). The bugs used in our experiments consumed 160 to 200 mg of blood. Only 15% of the insects did not feed fully and were discarded. The number of bugs dissected at different intervals after infection and the number of parasites per bug are summarized in Table I.
F, Villalta +3 more
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Modulation of NMDA Receptor Subunits Expression by Concanavalin A
Neurochemical Research, 2016The processes of N-methyl-D-aspartate (NMDA) receptor subunits expression were examined in cortical neurons and rat brain in order to investigate how the concanavalin A (Con A) modulates neuronal cells. Con A modulated the expression of NMDA receptor subunits in cultured cortical cells.
Soyong, Jang +3 more
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Mitogenic stimulation and the redistribution of concanavalin A receptors on lymphocytes
Experimental Cell Research, 1981Abstract When mouse spleen (Ig − ) cells undergo maximal mitogenic stimulation by optimal concentrations of concanavalin A (conA), the Ig − cells form caps of conA very slowly, with 50% of maximum cap formation occurring after about 10 h and maximal capping after about 24 h.
T, Pozzan +3 more
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Concanavalin A receptor capping in mouse myeloma and hybridoma
Cell Biology International Reports, 1986Concanavalin A capping was studied in immunoglobulin-secreting hybridomas derived from fusion of mouse myeloma NSO cells with mouse spleen lymphocytes. The cells of the parental populations differed significantly in capping ability (low in myeloma cells and high in the lymphocytes).
A, Staroselsky +4 more
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Mobility of Concanavalin a Receptors in Myelin and Synaptic Membranes
Nature New Biology, 1973THERE is now abundant evidence that biological membranes are fluid in nature with constituent protein and lipid molecules able to move relative to one another in the plane of the membrane1–6. In cells that occur in the unassociated state such as lymphocytes, surface macromolecules appear to be randomly distributed in the membrane unless cross linked by
A, Matus, S, De Petris, M C, Raff
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Microtubular Proteins and Concanavalin A Receptors
1975The inherent topographical distribution of Con A binding sites (CABS) is disperse or random in all cell types studied using hemocyanin to mark CABS in surface replicas. In virally transformed cells, the addition of Con A leads to the formation of clusters (CABS).
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