The Histamine Four Receptor Regulates LFA-1 But Not Mac-1 -Dependent Neutrophil Adhesion and Inflammatory Functions. [PDF]
Dib K.
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Formyl peptide receptor 2 activation by MR-39 inhibits glioblastoma cell proliferation and invasiveness through suppression of multiple oncogenic pathways. [PDF]
Ferraro MG +13 more
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On Vibronic-Driven Action and Mechanosensitive G Protein-Coupled Receptors. [PDF]
Cui ZJ, Huang WM, Li PJ, Xie XB.
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Porcine vomeronasal system: anatomy, development, receptor repertoire, and inflammatory change. [PDF]
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The Emerging Roles of Resolvins: Potential Diagnostic Biomarkers for Cardiovascular Diseases. [PDF]
Bolat R, Yazgan B.
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Targeting Neutrophil-Mediated Inflammation: Identification of Pyrazolidinone Carboxamide Derivatives as Potent Selective Inhibitors of Formyl Peptide Receptor 1 (FPR1)-Activated Neutrophils. [PDF]
Abdel-Halim M +10 more
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Mitochondria-derived peptides in liver disease: Emerging regulators of hepatic metabolism and therapeutic targets. [PDF]
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DAMPs cross-talk interpretation of MDD mechanisms. [PDF]
Peng Y, Chen Z, Xu Y, Ren B.
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The GPCR Connection: Linking Alzheimer's Disease and Glioblastoma. [PDF]
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Trends in Immunology, 2002Leukocytes accumulate at sites of inflammation and microbial infection in direct response to locally produced chemotactic factors, which signal through specific G protein-coupled receptors. The first chemotactic factors to be structurally defined were the N-formyl peptides. Unlike other leukocyte chemoattractants, N-formyl peptides could originate from
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