Results 141 to 150 of about 3,169 (190)
Regulation of osmolyte efflux by lysophospholipid receptors
, 2016 Heacock, A, Dodd, M, Fisher, S
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Biochimica Et Biophysica Acta - Molecular and Cell Biology of Lipids, 2004 Some of the biological effects of lipoproteins have been attributed to their association with lysophosphatidic acid (LPA), lysophosphatidylcholine (LPC), sphingosine-1-phosphate (S1P) and sphingosylphosphorylcholine (SPC). These lysophospholipids mediate multiple biological responses via several G protein-coupled receptors (GPR).
Christa Buechler, Gerd Schmitz
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Lysophospholipid Receptors: Signaling and Biology
Annual Review of Biochemistry, 2004 ▪ Abstract Lysophospholipids (LPs), such as lysophosphatidic acid and sphingosine 1-phosphate, are membrane-derived bioactive lipid mediators. LPs can affect fundamental cellular functions, which include proliferation, differentiation, survival, migration, adhesion, invasion, and morphogenesis.
Isao, Ishii +3 more
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International Union of Pharmacology. XXXIV. Lysophospholipid Receptor Nomenclature
Pharmacological Reviews, 2002 The lysophospholipids, lysophosphatidic acid (LPA) and sphingosine 1-phosphate (S1P), are now recognized as important extracellular signaling molecules. These lipid mediators are pleiotropic; among the most common cellular responses are mitogenesis, cell survival (anti-apoptosis), inhibition of adenylyl cyclase and calcium mobilization.
Chun, Jerold +7 more
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GPR55 and GPR35 and their relationship to cannabinoid and lysophospholipid receptors
Life Sciences, 2013This review presents a summary of what is known about the G-protein coupled receptors GPR35 and GPR55 and their potential characterization as lysophospholipid or cannabinoid receptors, respectively. Both GPR35 and GPR55 have been implicated as important targets in pain and cancer, and additional diseases as well.
Pingwei Zhao, Mary E Abood
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Lysophospholipid receptor nomenclature
Biochimica Et Biophysica Acta - Molecular and Cell Biology of Lipids, 2002Kevin R Lynch
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International Journal of Molecular Sciences, 2021 Activation of microglia and/or astrocytes often releases proinflammatory molecules as critical pathogenic mediators that can promote neuroinflammation and secondary brain damages in diverse diseases of the central nervous system (CNS).
Bhakta Prasad Gaire, Gaire Bhakta Prasad
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