Results 1 to 10 of about 141,217 (309)

A Novel SCNN1B Mutation in a Neonate With Systemic Pseudohypoaldosteronism Type 1: Case Report. [PDF]

open access: yesClin Case Rep
Early recognition of PHA1B in neonates with persistent hyperkalemia and hyponatremia is important for timely intervention. Genetic testing confirms the diagnosis, guiding long‐term management.
Bahadoran E, Saffari F, Moghbelinejad S.
europepmc   +2 more sources

The renal baroreflex: A systematic review and meta-analysis in healthy and hypertensive animals. [PDF]

open access: yesPhysiol Rep
The renal baroreflex describes the dose‐dependent relation between renal pressure and renin release. Former studies have approximated this relation through animal experiments, but the exact shape of the response curve and its alteration by hypertension ...
van Ochten M   +5 more
europepmc   +2 more sources

A case report of severe hypoaldosteronism following unilateral adrenalectomy for aldosteronoma [PDF]

open access: yesЭндокринная хирургия, 2020
Primary aldosteronism is characterized by hypertension and accounts for about 10% of hypertensive patients. Hyperkalemia and renal disease post adrenalectomy has been described in the literature.
Lilit Egshatyan
doaj   +1 more source

Primary Aldosteronism Screening Rates Differ with Sex, Race, and Comorbidities

open access: yesJournal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, 2022
Background Primary aldosteronism (PA) is a common but under‐recognized cause of secondary hypertension. Data directly comparing screening rates across single and overlapping indications are lacking. Methods and Results We conducted a retrospective review
Adina F. Turcu   +7 more
doaj   +1 more source

Olfactory receptor 78 modulates renin but not baseline blood pressure

open access: yesPhysiological Reports, 2021
Olfactory receptor 78 (Olfr78) is a G protein‐coupled receptor (GPCR) that is expressed in the juxtaglomerular apparatus (JGA) of the kidney as well as the peripheral vasculature, and is activated by gut microbial metabolites. We previously reported that
Brian G. Poll   +4 more
doaj   +1 more source

Renin Angiotensin System and Covid-19 Infection [PDF]

open access: yesAdvanced Pharmaceutical Bulletin, 2022
Purpose The new coronavirus, called SARS-CoV-2, is responsible for the recent global pandemic COVID-19. The status of the global pandemic COVID-19 is currently underway, and the virus has caused about 1.11 million deaths.
Francesco Ferrara, Antonio Vitiello
doaj   +1 more source

Hormonal and hemodynamic changes in middle-aged urban residents with obesity depending on blood pressure

open access: yesMìžnarodnij Endokrinologìčnij Žurnal, 2020
Background. The relevance of the study is due to the significant spread of overweight of varying severity among the population and a significant risk of developing comorbid pathological conditions, including arterial hypertension (AH).
E.V. Misyura   +2 more
doaj   +1 more source

Concurrent Primary Aldosteronism and Renal Artery Stenosis: An Overlooked Condition Inducing Resistant Hypertension

open access: yesFrontiers in Cardiovascular Medicine, 2022
To explore the clinical features of coexisting primary aldosteronism (PA) and renal artery stenosis (RAS), we retrospectively analyzed records from 71 patients with PA with RAS and a control group of 121 patients with PA without RAS. Aldosterone-to-renin
Lin Zhao   +8 more
doaj   +1 more source

Angiotensinogen and the Modulation of Blood Pressure

open access: yesFrontiers in Cardiovascular Medicine, 2021
The angiotensin peptides that control blood pressure are released from the non-inhibitory plasma serpin, angiotensinogen, on cleavage of its extended N-terminal tail by the specific aspartyl-protease, renin. Angiotensinogen had previously been assumed to
Zimei Shu   +4 more
doaj   +1 more source

Plasma Angiotensin II Is Increased in Critical Coronavirus Disease 2019

open access: yesFrontiers in Cardiovascular Medicine, 2022
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) employs angiotensin-converting enzyme 2 (ACE2) as its receptor for cell entrance, and studies have suggested that upon viral binding, ACE2 catalytic activity could be inhibited; therefore ...
Rafael L. Camargo   +20 more
doaj   +1 more source

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