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Critical Care Nursing Clinics of North America, 1990
A review of the mechanism of ischemic-reperfusion injury, proposed interventions to prevent injury, and future directions have been presented to enhance the practitioner's knowledge of this new, exciting concept in myocardial injury. There is increasing evidence in the literature that reperfusion injury may occur in other organ systems and is ...
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A review of the mechanism of ischemic-reperfusion injury, proposed interventions to prevent injury, and future directions have been presented to enhance the practitioner's knowledge of this new, exciting concept in myocardial injury. There is increasing evidence in the literature that reperfusion injury may occur in other organ systems and is ...
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Cytokines and reperfusion injury
Neurology, 1997There is increasing evidence that the inflammatory response plays an important role in the potentiation of central nervous system (CNS) ischemic injury. Much of this inflammatory response appears to be mediated by interleukins (IL), a multifunctional subclass of cytokines.
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Reperfused Myocardium: Stunning, Preconditioning, and Reperfusion Injury
1990In the late 1960s, when the deathtoll from myocardial infarction had caused a high level of public awareness, the National Institutes of Health, USA, decided to fund projects directly related to the experimental treatment of infarcts, to develop animal models best suited to study infarct size after coronary occlusion, and to develop quantitative ...
Wolfgang Schaper +2 more
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Pathophysiology of reperfusion injury
2011INTRODUCTION Ischaemia-Reperfusion Injury (IRI) is defined as the paradoxical exacerbation of cellular dysfunction and death, following restoration of blood flow to previously ischaemic tissues. Reestablishment of blood flow is essential to salvage ischaemic tissues.
Cowled, P., Fitridge, R.
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Chemokines in ischemia and reperfusion
Thrombosis and Haemostasis, 2007SummaryChemokine signaling plays an important role in the postischemic inflammatory response. Overlapping pathways involving reactive oxygen intermediates,Toll-like receptor (TLR) activation, the complement cascade and the nuclear factor (NF)- κ B system induce both CXC and CC chemokines in ischemic tissues. Reperfusion accentuates chemokine expression
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Cytokines and Reperfusion Injury
Journal of Cardiac Surgery, 1993Myocardial dysfunction following prolonged ischemia and reperfusion is at least partially dependent upon adhesion of neutrophils to myocardial and endothelial cells. Neutrophils are thought to contribute to reperfusion injury by two mechanisms: impairment of the microvasculature by physical obstruction, and secretion of products that damage ...
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Controlled Reperfusion Against Ischemia Reperfusion Injury
2018Recanalization and reperfusion after an ischemic stroke sometimes could be harmful and deadly, and controlled perfusion may be a potential solution. This article summarized controlled perfusion literature, both animal and clinical, on heart, lung, kidney, and limbs and compared with brain controlled perfusion.
Weijian Jiang +3 more
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Electrocardiographic Effects of Reperfusion
Cardiology Clinics, 1987This article focuses on the electrocardiographic events that may occur following restoration of flow in an infarct-related vessel.
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Therapeutic Coronary Reperfusion and Reperfusion Injury: An Introduction
Cardiology, 2016Jeffrey S, Borer, Basil S, Lewis
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