Editorial for the Special Issue "Molecular Mechanisms and Treatment of Ischemia-Reperfusion Injury". [PDF]
Kobayashi J.
europepmc +1 more source
Reperfusion injury in the age of revascularization [PDF]
David Dornbos III, Yuchuan Ding
openaire +3 more sources
Possible therapeutic repositioning of valproic acid: From epileptic seizures to acute kidney injury
Valproic acid, an anticonvulsant, may be repositioned to prevent acute kidney injury due to ischemia followed by reperfusion. It preserves renal functions, electrolyte homeostasis and active sodium transport in kidney tubules, and blocks the onset of hypertension.
Danilo Alves‐Bezerra +8 more
wiley +1 more source
MITF Regulates CFTR Expression to Participate in Myocardial Ischemia-Reperfusion Injury. [PDF]
Tang B, Zhu C, Wang H, Gao M, Li T.
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Multifaceted mechanisms of unconventional T cells in ischemia-reperfusion injury. [PDF]
Li J +10 more
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Atomically precise metal cluster enzymes for pathological tissue regeneration
Schematic illustration of atomically precise metal cluster enzymes (MCEs) for pathological tissue regeneration. Atomically precise MCEs can modulate biological processes, such as attenuation of inflammatory responses, eradication of bacterial pathogens, regulation of angiogenesis, and promotion of cell development.
Ziqiang Xiong +11 more
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Pathophysiological Effects and Targeted Therapy Strategies of Neutrophil Extracellular Traps in Ischemia-Reperfusion Injury. [PDF]
Lv Y +12 more
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Harnessing ferroptosis from multilayer defense networks to nanoplatforms for specific cancer therapy
Nanomaterials target metabolically‐regulated ferroptosis for cancer therapy. Iron‐based or alternative nanoplatforms integrate ferroptosis with chemotherapy, immunotherapy, or radiotherapy. They enable stimulus‐responsive therapies (photothermal, photodynamic, sonodynamic) activated by near‐infrared, light, or ultrasound, achieving potent synergistic ...
Xinyue Xu +5 more
wiley +1 more source
NP‐SF, designed to target macrophage Dectin‐1 and activated platelets, selectively accumulated in ischemic myocardium, where it was taken up by macrophages after platelet hitchhiking. By inhibiting NLRC5‐related PANoptosis, NP‐SF significantly attenuated pathological damage, and effectively preserved cardiac function in a murine ischemia–reperfusion ...
Guanye Yu +13 more
wiley +1 more source
METTL14 Promotes Cuproptosis-Associated Changes in Rats With Myocardial Ischemia-Reperfusion Injury. [PDF]
Zeng S, Hong D, Yang L, Wu Y.
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