Results 31 to 40 of about 1,633,210 (336)

The role of reactive oxygen species in antibiotic-induced cell death in Burkholderia cepacia complex bacteria [PDF]

open access: yes, 2016
It was recently proposed that bactericidal antibiotics, besides through specific drug-target interactions, kill bacteria by a common mechanism involving the production of reactive oxygen species (ROS).
Acke, Marloes   +5 more
core   +4 more sources

MT-12 inhibits the proliferation of bladder cells in vitro and in vivo by enhancing autophagy through mitochondrial dysfunction

open access: yesOpen Life Sciences, 2022
Bladder cancer (BC) is one of the most common malignancies involving the urinary system. Our previous study demonstrated that cobra venom membrane toxin 12 (MT-12) could effectively inhibit BC cell growth and metastasis and induce apoptosis. However, the
Chen Yan   +7 more
doaj   +1 more source

Mitochondria: Redox Metabolism and Dysfunction

open access: yesBiochemistry Research International, 2012
Mitochondria are the main intracellular location for fuel generation; however, they are not just power plants but involved in a range of other intracellular functions including regulation of redox homeostasis and cell fate.
Jia Kang, Shazib Pervaiz
doaj   +1 more source

Cu,Zn superoxide dismutase genes in Tribolium castaneum: evolution, molecular characterisation and gene expression during immune priming. [PDF]

open access: yes, 2017
The production of reactive oxygen species (ROS) is a normal consequence of the aerobic cell metabolism. Despite their high and potentially detrimental reactivity with various biomolecules, the endogenous production of ROS is a vital part of physiological,
Bakiu, Rigers   +5 more
core   +2 more sources

Involvement of Metabolic Lipid Mediators in the Regulation of Apoptosis

open access: yesBiomolecules, 2020
Apoptosis is the physiological mechanism of cell death and can be modulated by endogenous and exogenous factors, including stress and metabolic alterations.
Piotr Wójcik   +3 more
doaj   +1 more source

Syntaxin-17-Dependent Mitochondrial Dynamics is Essential for Protection Against Oxidative-Stress-Induced Apoptosis

open access: yesAntioxidants, 2019
In this study, cell death induced by the oxidant tert-butylhydroperoxide (tBH) was observed in U2OS cells; this phenotype was rescued by Syntaxin 17 (STX17) knockout (KO) but the mechanism is unknown.
Binran Wang   +3 more
doaj   +1 more source

Demonstration of a Validated Direct Current Wearable Device for Monitoring Sweat Rate in Sports

open access: yesSensors
Sweat rate magnitude is a desired outcome for any wearable sensing patch dedicated to sweat analysis. Indeed, sweat rate values can be used two-fold: self-diagnosis of dehydration and correction/normalization of other physiological metrics, such as Borg ...
Xing Xuan   +4 more
doaj   +1 more source

Intelligent SLA Selection Through the Validation Cloud Broker System

open access: yesIEEE Access
Cloud computing has transformed digital service delivery by providing scalable, flexible access to computing resources, including servers, storage, and applications, under a pay-per-use model.
Ihab Sekh, Karoly Nehez
doaj   +1 more source

Effect of LPS and melatonin on early development of mouse embryo

open access: yesJournal of Animal Reproduction and Biotechnology, 2022
Lipopolysaccharide (LPS) is an endotoxin factor present in the cell wall of Gram-negative bacteria and induces various immune responses to infection. Recent studies have reported that LPS induces cellular stress in various cells including oocytes and ...
Haeun Park, Hoon Jang, Youngsok Choi
doaj   +1 more source

Temperature and light requirements for growth of two diatom species (Bacillariophyceae) isolated from an Arctic macroalga [PDF]

open access: yes, 2006
In the present study, two abundant epiphyticdiatom taxa were isolated from the assimilation hairs ofthe brown macroalga Chordaria flagelliformis collected inthe Arctic Kongsfjorden (Spitsbergen, Norway), establishedas unialgal cultures and their growth ...
Jung, I.   +4 more
core   +1 more source

Home - About - Disclaimer - Privacy