Results 171 to 180 of about 140,542 (289)

TRIC-A Facilitates Sarcoplasmic Reticulum-Mitochondrial Ca<sup>2+</sup> Signaling Crosstalk in Cardiomyocytes. [PDF]

open access: yesCells
Li A   +11 more
europepmc   +1 more source

Primitive Phospholamban- and Sarcolipin-like Peptides Inhibit the Sarcoplasmic Reticulum Calcium Pump SERCA. [PDF]

open access: yesBiochemistry, 2022
Bak JJ   +8 more
europepmc   +1 more source

Caloric restriction and aerobic exercise differentially counteract muscle–tendon dysfunction following diet‐induced obesity in C57BL/6J mice

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Diet‐induced obesity induced fibro‐adipogenic remodelling of the muscle–tendon unit, characterized by increased fibrosis, reduced specific force, impaired relaxation kinetics, elevated hysteresis, and reduced aerobic capacity and glucose tolerance.
Leonardo Cesanelli   +9 more
wiley   +1 more source

Architecture of the cardiac transverse–axial tubular system across different mammalian species

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Mammalian species differ in ventricular TATS architecture, and cytoplasmic distance to the nearest TATS element covaries with lifespan, body mass, and resting heart rate. Abstract Cardiac excitation–contraction coupling relies on a pancellular network of regular cardiomyocyte surface membrane invaginations, termed the transverse ...
Joachim Greiner   +10 more
wiley   +1 more source

Alterations of sarcoplasmic reticulum-mediated Ca<sup>2+</sup> uptake in a model of premature ventricular contraction (PVC)-induced cardiomyopathy. [PDF]

open access: yesMol Cell Biochem, 2023
Balderas-Villalobos J   +9 more
europepmc   +1 more source

Mitochondrial permeability transition in skeletal muscle phenocopies muscle alterations seen in cancer cachexia and other wasting conditions

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Skeletal muscle wasting conditions are characterized by muscle fibre atrophy, mitochondrial respiratory dysfunction, mitochondrial depletion and fragmentation of acetylcholine receptor (AChR) cluster at the neuromuscular junction.
Maya Semel   +10 more
wiley   +1 more source

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