Results 11 to 20 of about 121,983 (190)
Rapid Field Immunoassay for Detecting Antibody to Sin Nombre Virus in Deer Mice [PDF]
Emerging Infectious Diseases, 2007 We developed a 1-hour field enzyme immunoassay (EIA) for detecting antibody to Sin Nombre virus in deer mice (Peromyscus maniculatus). The assay specificity and sensitivity were comparable to those of a standard EIA.
Tony Schountz +6 more
doaj +4 more sources
Sin nombre virus and rodent species diversity: a test of the dilution and amplification hypotheses. [PDF]
PLoS ONE, 2009 BACKGROUND:Species diversity is proposed to greatly impact the prevalence of pathogens. Two predominant hypotheses, the "Dilution Effect" and the "Amplification Effect", predict divergent outcomes with respect to the impact of species diversity.
Christine A Clay +3 more
doaj +5 more sources
Sin Nombre Virus Infection in Deer Mice, Channel Islands, California [PDF]
Emerging Infectious Diseases, 2008 To the Editor: Sin Nombre virus (SNV) is a highly virulent strain of hantavirus associated with rodent hosts in North America (1,2). Documenting the prevalence of SNV in wild rodent populations is an important component of determining risk for exposure and ultimately providing sound recommendations for epidemiologic management (3). Prevalence of SNV is
John L. Orrock, Brian F. Allan
doaj +4 more sources
Seroprevalence Against Sin Nombre Virus in Resident and Dispersing Deer Mice [PDF]
Vector-Borne and Zoonotic Diseases, 2008 Through dispersal, deer mice (Peromyscus maniculatus) enter peridomestic settings (e.g., outbuildings, barns, cabins) and expose humans and other deer mouse populations to Sin Nombre virus (SNV). In June 2004, research on deer mouse dispersal was initiated at 2 locations in Montana.
Lonner BN +3 more
exaly +5 more sources
Natural History of Sin Nombre Virus in Western Colorado
Emerging Infectious Diseases, 1999 A mark-recapture longitudinal study of immunoglobulin G (IgG) antibody to Sin Nombre virus (SNV) in rodent populations in western Colorado (1994—results summarized to October 1997) indicates the presence of SNV or a closely related hantavirus at two ...
Charles H. Calisher +3 more
doaj +3 more sources
Contact heterogeneity in deer mice: implications for Sin Nombre virus transmission [PDF]
Proceedings of the Royal Society B: Biological Sciences, 2009 Heterogeneities within disease hosts suggest that not all individuals have the same probability of transmitting disease or becoming infected. This heterogeneity is thought to be due to dissimilarity in susceptibility and exposure among hosts. As such, it has been proposed that many host–pathogen systems follow the general pattern whereby a
Maria Andrea Previtali, M Denise Dearing
exaly +5 more sources
Evaluating Neutralizing Antibodies in Hantavirus-Infected Patients Using Authentic Virus and Recombinant Vesicular Stomatitis Virus Systems [PDF]
VirusesHantaviruses, including the Sin Nombre virus (SNV) and Andes virus (ANDV), are associated with severe global health risks, causing high mortality rates in hantavirus pulmonary syndrome (HPS) patients.
Punya Shrivastava-Ranjan +10 more
doaj +2 more sources
A Longitudinal Study of Sin Nombre Virus Prevalence in Rodents, Southeastern Arizona
Emerging Infectious Diseases, 1999 We determined the prevalence of Sin Nombre virus antibodies in small mammals in southeastern Arizona. Of 1,234 rodents (from 13 species) captured each month from May through December 1995, only mice in the genus Peromyscus were seropositive.
Amy J. Kuenzi +4 more
doaj +3 more sources
Viruses, 2019 Andes virus (ANDV) and Sin Nombre virus (SNV) are the main causative agents responsible for hantavirus cardiopulmonary syndrome (HCPS) in the Americas.
Bryce M Warner +9 more
doaj +3 more sources
Removing Deer Mice from Buildings and the Risk for Human Exposure to Sin Nombre Virus
Emerging Infectious Diseases, 2003 Trapping and removing deer mice from ranch buildings resulted in an increased number of mice, including Sin Nombre virus antibody–positive mice, entering ranch buildings.
Richard J. Douglass +4 more
doaj +3 more sources