Results 211 to 220 of about 104,390 (258)
The Role of Matrix Metalloproteinases in Orthodontics, Dental Trauma, Restorative Dentistry, and Endodontics: Molecular Mechanisms and Clinical Implications. [PDF]
Int J Mol SciŁawicka R +6 more
europepmc +1 more source
The regenerative role of neural crest stem cells in physical stimuli-enhanced peripheral nerve repair. [PDF]
Stem Cell ReportsTai Y, Jin L, Tonmoy TI, Park BH, Nam J.
europepmc +1 more source
The development of intrapulmonary shunting caused by luspatercept in low grade myelodysplastic syndrome: a case report. [PDF]
AME Case RepFrisch A, Wilkinson JD, Nyquist AS.
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Decoding signaling crosstalk in pulpitis: pathogenesis and precision therapeutics. [PDF]
Front Cell Dev BiolJia Z, Sun Z, Zhou H.
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Phosphatases in SMAD regulation [PDF]
FEBS Letters, 2012 SMAD transcription factors are key mediators of the transforming growth factor‐beta (TGFß) family of cytokines. Reversible phosphorylation of SMAD proteins plays a key role in regulating their function. Several phosphatases have been proposed to act on SMAD proteins to influence TGFß/BMP signalling. Here we provide an overview of the SMAD regulation by
Sapkota G
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Cytokine & Growth Factor Reviews, 2000
Transforming growth factor-beta superfamily member signals are conveyed through cell-surface serine/threonine kinase receptors to the intracellular mediators known as Smads. Activation of Smads causes their translocation from the cytoplasm to the nucleus where they function to control gene expression.
J L, Wrana, L, Attisano
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Transforming growth factor-beta superfamily member signals are conveyed through cell-surface serine/threonine kinase receptors to the intracellular mediators known as Smads. Activation of Smads causes their translocation from the cytoplasm to the nucleus where they function to control gene expression.
J L, Wrana, L, Attisano
openaire +2 more sources
The International Journal of Biochemistry & Cell Biology, 1999
The Smads are a family of intracellular signalling molecules that act downstream of receptors for the transforming growth factor (TGF)-beta family of ligands. Three classes of Smads have been identified. The receptor-regulated Smads are direct substrates for the type I receptors, which are serine/threonine kinases.
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The Smads are a family of intracellular signalling molecules that act downstream of receptors for the transforming growth factor (TGF)-beta family of ligands. Three classes of Smads have been identified. The receptor-regulated Smads are direct substrates for the type I receptors, which are serine/threonine kinases.
openaire +2 more sources