Neutral Sphingomyelinase 2 (SMPD3) Deficiency in Mice Causes Chondrodysplasia with Unimpaired Skeletal Mineralization
Smpd3 Expression in both Chondrocytes and Osteoblasts Is Required for Normal Endochondral Bone Development
Mutations in the neutral sphingomyelinase gene SMPD3 implicate the ceramide pathway in human leukemias
TLR-Induced SMPD3 Defects Enhance Inflammatory Response of B Cell and Macrophage in the Pathogenesis of SLE
A neutron scattering investigation of the magnetic form factor for the intermultiplet transitions in Sm and SmPd3