Results 251 to 260 of about 1,032,066 (371)
An Off‐the‐Shelf Artificial Proregenerative Macrophage for Pressure Ulcer Treatment
Artificial macrophages (artM) by encapsulating M2 lysates into biodegradable PLGA microspheres and coated with macrophage membrane, are engineered as new therapeutics against skin pressure ulcer. In mouse models, the artM exhibited robust tissue repair through cell recruitment and microenvironment modulation, showing great therapeutic potential for ...
Qi Su+12 more
wiley +1 more source
Effect of Sucrose on the Rheology and 3D Printability of Pregelatinized Rice Flour Paste. [PDF]
Lee D, Saleh MI, Lee Y.
europepmc +1 more source
THE EFFECTS OF HIGH PRESSURE ON THE INVERSION OF SUCROSE AND THE MUTAROTATION OF GLUCOSE
Frank V. Sander
openalex +1 more source
Eosinophils play a crucial role in the progression of temporomandibular joint osteoarthritis (TMJOA). This study demonstrates that eosinophils, induced by OVA/IL‐5, promote Lumican secretion in the synovium, which binds to Annexin A1 in chondrocytes, inhibiting transforming growth factor β2 and Smad2/3 phosphorylation.
Wenqian Chen+12 more
wiley +1 more source
A Convenient Strategy for Studying Antibody Aggregation and Inhibition of Aggregation: Characterization and Simulation. [PDF]
Guo Y, Chen X, Fang G, Cao X, Wan J.
europepmc +1 more source
Intratracheal neomycin alters pulmonary microbiota, activating the vagus nerve via lung brain‐derived neurotrophic factor/tropomyosin receptor kinase B (BDNF/TrkB) pathway, further projecting to nucleus of solitary tract (NTS), then dorsal raphe nucleus (DRN), where activated serotoninergic neurons increase brain serotonin, alleviating migraine.
Biying Liu+7 more
wiley +1 more source
Breeding and regional production capacity performance of new sugarcane cultivar GT 66. [PDF]
Liang Q+12 more
europepmc +1 more source
Saturation relations in mixtures of sucrose, dextrose, and levulose.
R.F. Jackson, C.G. Silsbee
openalex +2 more sources
Under DNA damage, tumor cells rely on efficient DNA repair for survival and therapy resistance. This study has demonstrated that BCKDK localizes to breast cancer cell nuclei, where it binds to and phosphorylates RNF8, thereby blocking ubiquitin‐mediated degradation of RAD51 and enhancing HRR. A selective BCKDK inhibitor synergizes with clinical agents,
Haiying Liu+12 more
wiley +1 more source